Abstract
Nucleoside analog inhibitors (NAIs) are an important class of antiviral agents. Although highly effective, some NAIs with activity against hepatitis C virus (HCV) can cause toxicity, presumably due to off-target inhibition of host mitochondrial RNA polymerase (POLRMT). The in vitro nucleotide substrate specificity of POLRMT was studied in order to explore structure-activity relationships that can facilitate the identification of nontoxic NAIs. These findings have important implications for the development of all anti-RNA virus NAIs.
Keywords:
antiviral agents; hepatitis C virus; toxicity.
Copyright © 2017 American Society for Microbiology.
Publication types
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Research Support, Non-U.S. Gov't
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Research Support, N.I.H., Extramural
MeSH terms
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Amides / adverse effects
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Amides / pharmacology
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Antiviral Agents / adverse effects
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Antiviral Agents / pharmacology*
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Catalytic Domain / drug effects
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DNA-Directed RNA Polymerases / genetics*
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DNA-Directed RNA Polymerases / metabolism*
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Hepacivirus / drug effects*
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Hepatitis C / drug therapy*
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Humans
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Mitochondria / drug effects*
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Mitochondria / genetics
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Nucleosides / pharmacology
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Phosphoric Acids / adverse effects
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Phosphoric Acids / pharmacology
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Sofosbuvir / adverse effects
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Sofosbuvir / pharmacology
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Structure-Activity Relationship
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Substrate Specificity
Substances
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Amides
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Antiviral Agents
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Nucleosides
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Phosphoric Acids
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phosphoramidic acid
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DNA-Directed RNA Polymerases
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POLRMT protein, human
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Sofosbuvir