Abstract
Chromosome instability (CIN) contributes to the development of many cancer. In this paper, we summarize our recent finding that a novel pathway by which FBW7 loss promotes Centromere Protein A (CENP-A) phosphorylation on Serine 18 through Cyclin E1/CDK2, therefore promoting CIN and tumorigenesis. Our finding demonstrates the importance of CENP-A post-translational modification on modulating centromere and mitotic functions in cancer.
Keywords:
CENP-A; Cyclin E1; FBW7; centromere; chromosome instability; tumorigenesis.
Publication types
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Research Support, N.I.H., Extramural
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Research Support, Non-U.S. Gov't
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Research Support, U.S. Gov't, Non-P.H.S.
MeSH terms
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Carcinogenesis
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Centromere Protein A / genetics
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Centromere Protein A / metabolism*
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Chromosomal Instability*
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DNA-Binding Proteins / metabolism
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F-Box-WD Repeat-Containing Protein 7 / metabolism
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Humans
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Mutagenesis, Site-Directed
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Neoplasms / metabolism
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Neoplasms / pathology
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Phosphorylation
Substances
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Centromere Protein A
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DNA-Binding Proteins
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F-Box-WD Repeat-Containing Protein 7
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FBXW7 protein, human
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HJURP protein, human