BMAL1 functions as a cAMP-responsive coactivator of HDAC5 to regulate hepatic gluconeogenesis

Protein Cell. 2018 Nov;9(11):976-980. doi: 10.1007/s13238-018-0514-y.

Jian Li^{1

2}, Sihan Lv¹, Xinchen Qiu^{1

2}, Jiamin Yu¹, Junkun Jiang¹, Yalan Jin², Wenxuan Guo², Ruowei Zhao², Zhen-Ning Zhang³, Chao Zhang⁴, Bing Luan⁵

¹ Department of Endocrinology, Shanghai Tenth People's Hospital, School of Medicine, Tongji University, Shanghai, China.
² Translational Medical Center for Stem Cell Therapy & Institute for Regenerative Medicine, Shanghai East Hospital, Shanghai Key Laboratory of Signaling and Disease Research, School of Life Sciences and Technology, Tongji University, Shanghai, China.
³ Translational Medical Center for Stem Cell Therapy & Institute for Regenerative Medicine, Shanghai East Hospital, Shanghai Key Laboratory of Signaling and Disease Research, School of Life Sciences and Technology, Tongji University, Shanghai, China. znzhang@tongji.edu.cn.
⁴ Translational Medical Center for Stem Cell Therapy & Institute for Regenerative Medicine, Shanghai East Hospital, Shanghai Key Laboratory of Signaling and Disease Research, School of Life Sciences and Technology, Tongji University, Shanghai, China. zhangchao@tongji.edu.cn.
⁵ Department of Endocrinology, Shanghai Tenth People's Hospital, School of Medicine, Tongji University, Shanghai, China. bluan@tongji.edu.cn.

No abstract available

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