STK24 expression is modulated by DNA copy number/methylation in lung adenocarcinoma and predicts poor survival

Future Oncol. 2018 Sep;14(22):2253-2263. doi: 10.2217/fon-2018-0126. Epub 2018 Mar 20.

Abstract

Aim: To explore the independent prognostic value of STK24 expression in terms of overall survival and recurrence-free survival and the potential mechanisms of its dysregulation in non-small-cell lung adenocarcinoma.

Patients & methods: Data were from the Cancer Genome Atlas-lung adenocarcinoma.

Results: Increased STK24 expression was an independent prognostic indicator of unfavorable overall survival (Hazard ratio: 1.478; 95% CI: 1.149-1.901; p < 0.002) and recurrence-free survival (Hazard ratio: 1.855; 95% CI: 1.399-2.458; p < 0.001). DNA amplification was associated with significantly upregulated STK24 expression. There was a weak negative correlation between STK24 expression and its DNA methylation (Pearson's r = -0.32).

Conclusion: Aberrant STK24 expression was an independent prognostic indicator in lung adenocarcinoma patients. Its dysregulation was associated with its DNA copy number alteration and methylation.

Keywords: STK24; copy number; lung adenocarcinoma; methylation; prognosis.

MeSH terms

  • Adenocarcinoma of Lung / genetics*
  • Adenocarcinoma of Lung / metabolism
  • Adenocarcinoma of Lung / mortality*
  • Aged
  • Biomarkers, Tumor / genetics
  • Biomarkers, Tumor / metabolism
  • Case-Control Studies
  • DNA Copy Number Variations*
  • DNA Methylation*
  • Disease-Free Survival
  • Female
  • Humans
  • Kaplan-Meier Estimate
  • Male
  • Middle Aged
  • Prognosis
  • Protein Serine-Threonine Kinases / genetics
  • Protein Serine-Threonine Kinases / metabolism*
  • Up-Regulation

Substances

  • Biomarkers, Tumor
  • STK24 protein, human
  • Protein Serine-Threonine Kinases