Nonsynonymous TNNI3K mutations and phenotypes: More than a "simple" cardiomyopathy

Heart Rhythm. 2019 Jan;16(1):106-107. doi: 10.1016/j.hrthm.2018.07.033. Epub 2018 Jul 29.

Authors

Sven Dittmann¹, Eric Schulze-Bahr²

Affiliations

¹ Institute for Genetics of Heart Diseases (IfGH), University Hospital Münster, Münster, Germany.
² Institute for Genetics of Heart Diseases (IfGH), University Hospital Münster, Münster, Germany. Electronic address: eric.schulze-bahr@ukmuenster.de.

PMID: 30063992
DOI: 10.1016/j.hrthm.2018.07.033

No abstract available

Publication types

Editorial
Comment

MeSH terms

Cardiac Conduction System Disease
Cardiomyopathies*
Humans
MAP Kinase Kinase Kinases
Mutation
Phenotype
Protein Serine-Threonine Kinases
Tachycardia, Supraventricular*

Substances

Protein Serine-Threonine Kinases
TNNI3K protein, human
MAP Kinase Kinase Kinases