Islet-Like Structures Generated In Vitro from Adult Human Liver Stem Cells Revert Hyperglycemia in Diabetic SCID Mice

Stem Cell Rev Rep. 2019 Feb;15(1):93-111. doi: 10.1007/s12015-018-9845-6.

Abstract

A potential therapeutic strategy for diabetes is the transplantation of induced-insulin secreting cells. Based on the common embryonic origin of liver and pancreas, we studied the potential of adult human liver stem-like cells (HLSC) to generate in vitro insulin-producing 3D spheroid structures (HLSC-ILS). HLSC-ILS were generated by a one-step protocol based on charge dependent aggregation of HLSC induced by protamine. 3D aggregation promoted the spontaneous differentiation into cells expressing insulin and several key markers of pancreatic β cells. HLSC-ILS showed endocrine granules similar to those seen in human β cells. In static and dynamic in vitro conditions, such structures produced C-peptide after stimulation with high glucose. HLSC-ILS significantly reduced hyperglycemia and restored a normo-glycemic profile when implanted in streptozotocin-diabetic SCID mice. Diabetic mice expressed human C-peptide and very low or undetectable levels of murine C-peptide. Hyperglycemia and a diabetic profile were restored after HLSC-ISL explant. The gene expression profile of in vitro generated HLSC-ILS showed a differentiation from HLSC profile and an endocrine commitment with the enhanced expression of several markers of β cell differentiation. The comparative analysis of gene expression profiles after 2 and 4 weeks of in vivo implantation showed a further β-cell differentiation, with a genetic profile still immature but closer to that of human islets. In conclusion, protamine-induced spheroid aggregation of HLSC triggers a spontaneous differentiation to an endocrine phenotype. Although the in vitro differentiated HLSC-ILS were immature, they responded to high glucose with insulin secretion and in vivo reversed hyperglycemia in diabetic SCID mice.

Keywords: 3D culture; Diabetes; Insulin-producing stem cells; Liver stem cells; Pancreatic islets; Pancreatic β cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Animals
  • Biomarkers / metabolism
  • C-Peptide / metabolism
  • Cell Differentiation / drug effects
  • Diabetes Mellitus, Experimental / complications*
  • Diabetes Mellitus, Experimental / therapy*
  • Gene Expression Regulation / drug effects
  • Glucose / pharmacology
  • Humans
  • Hyperglycemia / complications*
  • Hyperglycemia / therapy*
  • Insulin-Secreting Cells / drug effects
  • Insulin-Secreting Cells / metabolism
  • Islets of Langerhans / physiology*
  • Islets of Langerhans / ultrastructure
  • Liver / cytology*
  • Male
  • Mice, SCID
  • Phenotype
  • Protamines / pharmacology
  • Spheroids, Cellular / cytology
  • Spheroids, Cellular / drug effects
  • Stem Cells / cytology*
  • Stem Cells / drug effects

Substances

  • Biomarkers
  • C-Peptide
  • Protamines
  • Glucose