Itraconazole perturbs colorectal cancer dormancy through SUFU-mediated WNT inhibition

Mol Cell Oncol. 2018 Aug 7;5(4):e1494950. doi: 10.1080/23723556.2018.1494950. eCollection 2018.

Abstract

Cancer cell dormancy is an important source of treatment failure. We studied the molecular characteristics and functional behaviour of dormant colorectal cancer cells finding them to be a differentiated yet plastic population. Organoid drug screening identified itraconazole perturbs dormancy through non-canonical hedgehog signalling effects on the WNT pathway.

Keywords: cancer stem cell; cell cycle; colorectal cancer; dormancy; hedgehog; heterogeneity; itraconazole; plasticity; wnt.