RalB directly triggers invasion downstream Ras by mobilizing the Wave complex

Elife. 2018 Oct 15:7:e40474. doi: 10.7554/eLife.40474.

Abstract

The two Ral GTPases, RalA and RalB, have crucial roles downstream Ras oncoproteins in human cancers; in particular, RalB is involved in invasion and metastasis. However, therapies targeting Ral signalling are not available yet. By a novel optogenetic approach, we found that light-controlled activation of Ral at plasma-membrane promotes the recruitment of the Wave Regulatory Complex (WRC) via its effector exocyst, with consequent induction of protrusions and invasion. We show that active Ras signals to RalB via two RalGEFs (Guanine nucleotide Exchange Factors), RGL1 and RGL2, to foster invasiveness; RalB contribution appears to be more important than that of MAPK and PI3K pathways. Moreover, on the clinical side, we uncovered a potential role of RalB in human breast cancers by determining that RalB expression at protein level increases in a manner consistent with progression toward metastasis. This work highlights the Ras-RGL1/2-RalB-exocyst-WRC axis as appealing target for novel anticancer strategies.

Keywords: Exocyst; Invasion; Ral; Wave; breast cancer; cell biology; human; optogenetics.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Breast Neoplasms / metabolism
  • Breast Neoplasms / pathology
  • Cell Line
  • Cell Membrane / metabolism
  • Cell Membrane / radiation effects
  • Cell Surface Extensions / metabolism
  • Cell Surface Extensions / radiation effects
  • Disease Progression
  • Female
  • Humans
  • Light
  • Neoplasm Invasiveness
  • Optogenetics
  • Signal Transduction
  • Wiskott-Aldrich Syndrome Protein Family / metabolism*
  • ral GTP-Binding Proteins / metabolism*
  • ras Proteins / metabolism*

Substances

  • Ralb protein, human
  • Wiskott-Aldrich Syndrome Protein Family
  • ral GTP-Binding Proteins
  • ras Proteins

Grants and funding

The funders had no role in study design, data collection and interpretation, or the decision to submit the work for publication.