Anti-CAR-engineered T cells for epitope-based elimination of autologous CAR T cells

Cancer Immunol Immunother. 2019 Sep;68(9):1401-1415. doi: 10.1007/s00262-019-02376-y. Epub 2019 Aug 14.

Abstract

Although CAR T-cell therapy has demonstrated tremendous clinical efficacy especially in hematological malignancies, severe treatment-associated toxicities still compromise the widespread application of this innovative technology. Therefore, developing novel approaches to abrogate CAR T-cell-mediated side effects is of great relevance. Several promising strategies pursue the selective antibody-based depletion of adoptively transferred T cells via elimination markers. However, given the limited half-life and tissue penetration, dependence on the patients' immune system and on-target/off-side effects of proposed monoclonal antibodies, we sought to exploit αCAR-engineered T cells to efficiently eliminate CAR T cells. For comprehensive and specific recognition, a small peptide epitope (E-tag) was incorporated into the extracellular spacer region of CAR constructs. We provide first proof-of-concept for targeting this epitope by αE-tag CAR T cells, allowing an effective killing of autologous E-tagged CAR T cells both in vitro and in vivo whilst sparing cells lacking the E-tag. In addition to CAR T-cell cytotoxicity, the αE-tag-specific T cells can be empowered with cancer-fighting ability in case of relapse, hence, have versatile utility. Our proposed methodology can most probably be implemented in CAR T-cell therapies regardless of the targeted tumor antigen aiding in improving overall safety and survival control of highly potent gene-modified cells.

Keywords: Chimeric antigen receptor; Elimination tag; Immunotherapy; Toxicity management.

MeSH terms

  • Animals
  • Antigens, Neoplasm / genetics
  • Antigens, Neoplasm / immunology
  • Autoantigens / immunology
  • Cytotoxicity, Immunologic
  • Epitopes, T-Lymphocyte / genetics*
  • Epitopes, T-Lymphocyte / immunology
  • Genetic Engineering
  • Humans
  • Immunotherapy, Adoptive / methods*
  • Male
  • Mice
  • Neoplasm Recurrence, Local
  • PC-3 Cells
  • Peptide Fragments / genetics*
  • Prostatic Neoplasms / immunology
  • Prostatic Neoplasms / therapy*
  • Receptors, Antigen, T-Cell / genetics*
  • Receptors, Chimeric Antigen / genetics*
  • T-Lymphocytes, Cytotoxic / immunology*
  • Xenograft Model Antitumor Assays

Substances

  • Antigens, Neoplasm
  • Autoantigens
  • Epitopes, T-Lymphocyte
  • Peptide Fragments
  • Receptors, Antigen, T-Cell
  • Receptors, Chimeric Antigen