Dorsal hippocampal changes in T2 relaxation times are associated with early spatial cognitive deficits in 5XFAD mice

Brain Res Bull. 2019 Nov:153:150-161. doi: 10.1016/j.brainresbull.2019.08.011. Epub 2019 Aug 15.

Abstract

T2 relaxation time (T2) alterations may serve as markers for early detection and disease progression monitoring by reflecting brain microstructural integrity in Alzheimer's disease (AD). However, the characteristics of T2 alterations during the early stage of AD remain elusive. We explored T2 alterations and their possible correlations with cognitive function in 5XFAD mice at early ages (1, 2, 3, and 5 months of age). Voxel-based analysis (VBA) and region of interest (ROI) analysis showed a decreased T2 in the hippocampus of 2-, 3-, and 5-month-old 5XFAD mice compared to those of controls. The dorsal hippocampal T2 decreased earlier than the ventral hippocampus T2. A significant correlation was observed between Morris water maze (MWM) test cognitive behavior and the dorsal hippocampus T2 in 5XFAD mice. These results indicated that the microstructural integrity of brain tissues, particularly the hippocampus, was impaired early and the impairment became more extensive and severe during disease progression. Furthermore, the dorsal hippocampus is a crucial component involved in spatial cognition impairment in young 5XFAD mice.

Keywords: Alzheimer's disease; Cognition; Hippocampus; Magnetic resonance imaging; T2 relaxation time; Transgenic mouse model.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aging
  • Alzheimer Disease / diagnostic imaging*
  • Alzheimer Disease / metabolism
  • Alzheimer Disease / physiopathology
  • Amyloid beta-Protein Precursor / metabolism
  • Animals
  • Brain / metabolism
  • Cognition / physiology
  • Cognition Disorders / physiopathology
  • Cognitive Dysfunction / physiopathology
  • Diffusion Magnetic Resonance Imaging / methods*
  • Disease Models, Animal
  • Disease Progression
  • Hippocampus / metabolism
  • Humans
  • Male
  • Memory Disorders / physiopathology
  • Mice
  • Mice, Inbred C57BL
  • Mice, Transgenic
  • Presenilin-1 / metabolism
  • Spatial Navigation / physiology
  • Temporal Lobe / metabolism

Substances

  • Amyloid beta-Protein Precursor
  • Presenilin-1