Abstract
In this issue of Blood, Margraf et al selectively delete integrin linked kinase (ILK) in myeloid cells of mice to show that this integrin-binding protein suppresses chemokine-induced neutrophil extravasation and ischemia-induced reperfusion injury.
MeSH terms
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CD18 Antigens*
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Chemokines
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Humans
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Inflammation
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Lymphocyte Function-Associated Antigen-1*
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Neutrophils
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Protein Serine-Threonine Kinases
Substances
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CD18 Antigens
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Chemokines
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Lymphocyte Function-Associated Antigen-1
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integrin-linked kinase
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Protein Serine-Threonine Kinases