Bu-Shen-Zhu-Yun decoction induces PRLR deubiquitination and JAK2/STAT5 activation via CSN5 in vitro

Aging (Albany NY). 2021 Aug 23;13(16):20418-20437. doi: 10.18632/aging.203426. Epub 2021 Aug 23.

Abstract

Purpose: To determine the effect of Bu-Shen-Zhu-Yun Decoction (BSZY-D) on the kisspeptin through JAK2/STAT5 signaling pathway in hyperprolactinemia (HPRL) infertility.

Method: SD rats were treated with BSZY-D for cerebrospinal fluid (CSF) extraction. GT1-7 cells were subjected to different treatments. The phosphorylation levels of JAK2 and STAT5, and the expressions of PRLR and kisspeptin of GT1-7 cells in different groups were detected by western blot, RT-qPCR and immunofluorescence. The expressions of CSN5 and GATA1 and other molecular features were checked by western blot, RT-PCR, co-immunoprecipitation and renilla luciferase activity.

Results: The phosphorylation levels of JAK2 and STAT5, and the expressions of PRLR and kisspeptin in the HPRL group were significantly decreased, and these changes could be reversed after BSZY-D treatment. In addition, the presence of PRLR deubiquitination was detected in the HPRL group, which could be reversed by shRNA-CSN5, suggesting that BSZY-D played a role through targeting CSN5. The binding level of GATA1 and CSN5 promoter in the HPRL group was significantly decreased, but elevated in the HPRL (BSZY-D/CSF) group (P < 0.05).

Conclusion: BSZY-D improved the transcription activity of GATA1 and increased the binding of GATA1 and CSN5. BSZY-D was involved in the deubiquitination of PRLR, which contributes to alleviating the symptoms of HPRL infertility.

Keywords: Bu-Shen-Zhu-Yun decoction; hyperprolactinemia infertility.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • COP9 Signalosome Complex / genetics
  • COP9 Signalosome Complex / metabolism*
  • Drugs, Chinese Herbal / administration & dosage*
  • Female
  • GATA1 Transcription Factor / genetics
  • GATA1 Transcription Factor / metabolism
  • Humans
  • Hyperprolactinemia / drug therapy*
  • Hyperprolactinemia / genetics
  • Hyperprolactinemia / metabolism
  • Intracellular Signaling Peptides and Proteins / genetics
  • Intracellular Signaling Peptides and Proteins / metabolism*
  • Janus Kinase 2 / genetics
  • Janus Kinase 2 / metabolism*
  • Prolactin / genetics
  • Prolactin / metabolism*
  • Rats
  • Rats, Sprague-Dawley
  • STAT5 Transcription Factor / genetics
  • STAT5 Transcription Factor / metabolism*
  • Signal Transduction / drug effects

Substances

  • Drugs, Chinese Herbal
  • GATA1 Transcription Factor
  • Gata1 protein, rat
  • Gps1 protein, rat
  • Intracellular Signaling Peptides and Proteins
  • STAT5 Transcription Factor
  • Prolactin
  • Jak2 protein, rat
  • Janus Kinase 2
  • COP9 Signalosome Complex