The effects of biologic agents on cardiovascular risk factors and atherosclerosis in rheumatoid arthritis patients: a prospective observational study

Heart Vessels. 2022 Dec;37(12):2128-2136. doi: 10.1007/s00380-022-02114-y. Epub 2022 Jun 24.

Abstract

We aimed to evaluate the impact of biologic treatment on subclinical atherosclerosis and risk factors for cardiovascular disease (CVD) in patients with rheumatoid arthritis (RA). Forty-nine biologic naïve RA patients, treated with conventional synthetic disease-modifying anti-rheumatic drugs (csDMARDs), who were eligible for treatment with a biologic agent, were included in the study. The serum levels of lipid parameters, as well as disease activity parameters were determined in RA patients before and after 3 and 6 months of therapy. Carotid artery intima-media thickness (cIMT) was measured before and after treatment. A comparison analysis of change of these parameters was also performed between anti-tumor necrosis factor (anti-TNF) and non-anti-TNF users. Furthermore, 31 non-smoking healthy volunteers, matched for age and gender, were used as a control group. At baseline, RA patients had a decrease in serum total cholesterol (TC) and high-density lipoprotein cholesterol (HDL-C) levels compared with controls (209 ± 63 vs 233 ± 44 and 58 ± 15 vs 61 ± 14, p < 0.004), while cIMT was higher versus controls [0.9 (0.8-1) vs 0.6 (0.5-0.7), p < 0.001]. TC, HDL-C and apolipoprotein A1 levels were significantly increased 3 months after treatment (209 ± 63, 58 ± 15, 162 ± 32, vs 227 ± 45, 60 ± 15, 169 ± 29, respectively, p < 0.03) and this observation remained stable at a 6-month follow-up. After 6 months, there was also a statistically significant decrease in the cIMT [0.9 (0.8-1) vs 0.7 (0.6-0.8), p < 0.001]. Anti-TNF and non-anti-TNF users had comparable changes in cardiovascular risk parameters. The atherogenic lipid profile and subclinical atherosclerosis are features of RA, which appeared improved after biologic therapy initiation.

Keywords: Biologic agents; Cardiovascular risk; Intima-media thickness; Lipid profile; Rheumatoid arthritis; Subclinical atherosclerosis.

Publication types

  • Observational Study

MeSH terms

  • Antirheumatic Agents* / therapeutic use
  • Apolipoprotein A-I / therapeutic use
  • Arthritis, Rheumatoid* / complications
  • Arthritis, Rheumatoid* / diagnosis
  • Arthritis, Rheumatoid* / drug therapy
  • Atherosclerosis* / diagnosis
  • Atherosclerosis* / drug therapy
  • Atherosclerosis* / etiology
  • Biological Products* / pharmacology
  • Cardiovascular Diseases* / diagnosis
  • Cardiovascular Diseases* / etiology
  • Cardiovascular Diseases* / prevention & control
  • Carotid Intima-Media Thickness
  • Cholesterol
  • Heart Disease Risk Factors
  • Humans
  • Lipoproteins, HDL
  • Risk Factors
  • Tumor Necrosis Factor-alpha

Substances

  • Antirheumatic Agents
  • Apolipoprotein A-I
  • Biological Products
  • Cholesterol
  • Lipoproteins, HDL
  • Tumor Necrosis Factor-alpha