Polymeric micelles effectively reprogram the tumor microenvironment to potentiate nano-immunotherapy in mouse breast cancer models

Nat Commun. 2022 Nov 22;13(1):7165. doi: 10.1038/s41467-022-34744-1.

Abstract

Nano-immunotherapy improves breast cancer outcomes but not all patients respond and none are cured. To improve efficacy, research focuses on drugs that reprogram cancer-associated fibroblasts (CAFs) to improve therapeutic delivery and immunostimulation. These drugs, however, have a narrow therapeutic window and cause adverse effects. Developing strategies that increase CAF-reprogramming while limiting adverse effects is urgent. Here, taking advantage of the CAF-reprogramming capabilities of tranilast, we developed tranilast-loaded micelles. Strikingly, a 100-fold reduced dose of tranilast-micelles induces superior reprogramming compared to free drug owing to enhanced intratumoral accumulation and cancer-associated fibroblast uptake. Combination of tranilast-micelles and epirubicin-micelles or Doxil with immunotherapy increases T-cell infiltration, resulting in cures and immunological memory in mice bearing immunotherapy-resistant breast cancer. Furthermore, shear wave elastography (SWE) is able to monitor reduced tumor stiffness caused by tranilast-micelles and predict response to nano-immunotherapy. Micellar encapsulation is a promising strategy for TME-reprogramming and SWE is a potential biomarker of response.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Drug-Related Side Effects and Adverse Reactions*
  • Immunologic Factors
  • Immunotherapy
  • Mice
  • Micelles
  • Neoplasms*
  • Polymers
  • Tumor Microenvironment
  • ortho-Aminobenzoates / pharmacology
  • ortho-Aminobenzoates / therapeutic use

Substances

  • Micelles
  • tranilast
  • ortho-Aminobenzoates
  • Immunologic Factors
  • Polymers