An improved method reveals the dual role of Tnni3k in promoting S-phase entry while suppressing cell division in cardiomyocytes

J Mol Cell Cardiol. 2023 Oct:183:100-101. doi: 10.1016/j.yjmcc.2023.09.007. Epub 2023 Sep 26.

Authors

Lin Liu¹, Rich Gang Li¹, James F Martin²

Affiliations

¹ Cardiomyocyte Renewal Laboratory, Texas Heart Institute, 6770 Bertner Avenue, Houston, TX 77030, USA.
² Cardiomyocyte Renewal Laboratory, Texas Heart Institute, 6770 Bertner Avenue, Houston, TX 77030, USA; Department of Molecular Physiology and Biophysics, Baylor College of Medicine, One Baylor Plaza, Houston, TX 77030, USA. Electronic address: jfmartin@bcm.edu.

PMID: 37769962
DOI: 10.1016/j.yjmcc.2023.09.007

No abstract available

Publication types

Editorial

MeSH terms

Cell Division
MAP Kinase Kinase Kinases / genetics
Myocytes, Cardiac*
Protein Serine-Threonine Kinases*

Substances

Protein Serine-Threonine Kinases
MAP Kinase Kinase Kinases