Transient second-degree type 2 atrioventricular block after infliximab infusion in a patient with Crohn's disease and heterozygous familial hypercholesterolemia

Rev Esp Enferm Dig. 2024 Apr;116(4):233-234. doi: 10.17235/reed.2023.9950/2023.

Abstract

Current treatments for patients in the active phase of Crohn's disease (CD) include conventional treatments and biological treatments. Infliximab (IFX), a TNF-α antagonist, is recommended to induce remission in patients with moderate-to-severe CD who have not responded to conventional therapy. IFX terminates the inflammatory cascade by inhibiting the nuclear factor-κB (NF-κB), mitogen-activated protein kinase (MAPK), and caspase signaling pathways and increases the apoptosis of activated T cells in inflamed tissues.

MeSH terms

  • Antibodies, Monoclonal / therapeutic use
  • Atrioventricular Block* / chemically induced
  • Crohn Disease* / chemically induced
  • Gastrointestinal Agents / adverse effects
  • Humans
  • Infliximab / adverse effects
  • Treatment Outcome
  • Tumor Necrosis Factor-alpha

Substances

  • Infliximab
  • Antibodies, Monoclonal
  • Tumor Necrosis Factor-alpha
  • Gastrointestinal Agents