A reproducible murine model of studying HIV-associated brain damage in stroke

Brain Res. 2025 Jan 1:1846:149256. doi: 10.1016/j.brainres.2024.149256. Epub 2024 Oct 2.

Abstract

Background: Emerging clinical and epidemiological data indicates that human immunodeficiency virus (HIV) is associated with an increased risk of stroke and aggravated brain damage. We aimed to develop a reproducible murine model of photothrombotic-stroke with HIV infection that mimics the clinical situation.

Method: To evaluate the impact of HIV infection on stroke, male C57BL/6 mice were infected with EcoHIV (p24 2-4 × 106/mouse; i.v.) or mock control. Four weeks post-infection, a stroke was induced by the photothrombotic method (pt-MCAO). After 72 h, a catwalk test was performed for gait impairments, and mice were euthanized for stroke outcomes.

Results: EcoHIV-infection exhibited a larger infarction, brain edema, higher IgG extravasation, hemorrhagic transformation, and gait impairments following pt-MCAO vs mock control. EcoHIV-infected mice showed higher levels of IFN-y and lower levels of IL-6, indicating immune activation without affecting IL-1β and MCP-1 in plasma and brain compared to mock pt-MCAO, suggesting unaltered inflammation. EcoHIV-infection showed increased oxidative stress markers (nitrotyrosine, and 4-hydroxynonenal) and thioredoxin interacting protein expression. Further, EcoHIV-infection significantly activated the microglia and astrocyte cells.

Conclusions: This animal model would be reliable and clinically relevant to future studies investigating pathophysiological mechanisms and developing new therapeutic approaches in stroke patients with HIV conditions.

Keywords: Brain damage; EcoHIV-infection; Infract volume; Photothrombotic stroke.

MeSH terms

  • Animals
  • Brain / metabolism
  • Brain / pathology
  • Brain Injuries / pathology
  • Disease Models, Animal*
  • HIV Infections* / complications
  • Male
  • Mice
  • Mice, Inbred C57BL*
  • Oxidative Stress / physiology
  • Stroke* / pathology