A hTfR1 Receptor-Specific VHH Antibody Neutralizes Pseudoviruses Expressing Glycoproteins from Junín and Machupo Viruses

Viruses. 2024 Dec 20;16(12):1951. doi: 10.3390/v16121951.

Abstract

The Junín virus (JUNV) is one of the New World arenaviruses that cause severe hemorrhagic fever. Human transferrin receptor 1 (hTfR1) has been identified as the main receptor for JUNV for virus entry into host cells. To date, no treatment has been approved for JUNV. Herein, we investigated 12 anti-hTfR1 VHH (variable domain of the heavy chain of heavy-chain antibody) antibodies and confirmed their interaction with hTfR1. Most of them could bind to the hTfR1 apical domain, which is the glycoprotein 1 (GP1) binding domain of JUNV. Among them, 18N18 exhibited neutralizing activity against both the human immunodeficiency virus (HIV)-vectored lentiviral Junín pseudoviruses and the recombinant vesicular stomatitis virus (VSV)-vectored Junín pseudoviruses. We also verified that 18N18 blocked the interaction between hTfR1 and JUNV GP1. In addition, 18N18 could neutralize another New World arenavirus, the Machupo virus. Using AlphaFold 3-based simulation of 18N18-hTfR1 docking, we determined that 18N18's binding epitope was located at the JUNV GP1 binding epitope. 18N18 represents a candidate for JUNV treatment and provides a potential approach that could be applied to New World arenaviruses.

Keywords: Junín viruses; Machupo viruses; VHH antibody; transferrin receptor 1.

MeSH terms

  • Animals
  • Antibodies, Neutralizing* / immunology
  • Antibodies, Viral / immunology
  • Antigens, CD / genetics
  • Antigens, CD / immunology
  • Antigens, CD / metabolism
  • Arenaviruses, New World / genetics
  • Arenaviruses, New World / immunology
  • Epitopes / immunology
  • Glycoproteins* / genetics
  • Glycoproteins* / immunology
  • Glycoproteins* / metabolism
  • HEK293 Cells
  • Humans
  • Junin virus* / genetics
  • Junin virus* / immunology
  • Receptors, Transferrin* / immunology
  • Receptors, Transferrin* / metabolism
  • Receptors, Virus / immunology
  • Receptors, Virus / metabolism
  • Single-Domain Antibodies / genetics
  • Single-Domain Antibodies / immunology
  • Single-Domain Antibodies / metabolism
  • Viral Envelope Proteins / genetics
  • Viral Envelope Proteins / immunology
  • Viral Envelope Proteins / metabolism
  • Virus Internalization

Substances

  • Antibodies, Neutralizing
  • Glycoproteins
  • Receptors, Transferrin
  • CD71 antigen
  • Antibodies, Viral
  • Epitopes
  • Antigens, CD
  • Receptors, Virus
  • Viral Envelope Proteins
  • Single-Domain Antibodies

Grants and funding

This research received no external funding.