Adropin/Tirzepatide Combination Mitigates Cardiac Metabolic Aberrations in a Rat Model of Polycystic Ovarian Syndrome, Implicating the Role of the AKT/GSK3β/NF-κB/NLRP3 Pathway

Int J Mol Sci. 2024 Dec 24;26(1):1. doi: 10.3390/ijms26010001.

Abstract

Polycystic ovarian syndrome (PCOS) is a multifaceted metabolic and hormonal disorder in females of reproductive age, frequently associated with cardiac disturbances. This research aimed to explore the protective potential of adropin and/or tirzepatide (Tirze) on cardiometabolic aberrations in the letrozole-induced PCOS model. Female Wistar non-pregnant rats were allotted into five groups: CON; PCOS; PCOS + adropin; PCOS + Tirze; and PCOS + adropin+ Tirze. The serum sex hormones, glucose, and lipid profiles were securitized. Cardiac phosphorylated levels of AKT(pAKT), glycogen synthase kinase-3 beta (pGSK-3β), NOD-like receptor family pyrin domain containing 3 (NLPR3), IL-1β and IL-18 were assayed. The cardiac redox status and endoplasmic reticulum stress (ER) parameters including relative glucose-regulated protein 78 (GRP78) and C/EBP homologous protein (CHOP) gene expressions were detected. Finally, the immunoreactivity of cardiac NF-κB, Bcl2, and BAX were assessed. Our results displayed that adropin and/or Tirze intervention successfully alleviated the PCOS-provoked cardiometabolic derangements with better results recorded for the combination treatment. The synergistic effect of adropin and Tirze is mostly mediated via activating the cardiac Akt, which dampens the GSK3β/NF-κB/NLRP3 signaling pathway, with a sequel of alleviating oxidative damage, inflammatory response, ER stress, and related apoptosis, making them alluring desirable therapeutic targets in PCOS-associated cardiac complications.

Keywords: adropin; apoptosis; cardiovascular; inflammation; polycystic ovarian syndrome; tirzepatide.

MeSH terms

  • Animals
  • Blood Proteins
  • Disease Models, Animal*
  • Endoplasmic Reticulum Stress / drug effects
  • Female
  • Glycogen Synthase Kinase 3 beta* / metabolism
  • Intercellular Signaling Peptides and Proteins / metabolism
  • NF-kappa B* / metabolism
  • NLR Family, Pyrin Domain-Containing 3 Protein* / metabolism
  • Peptide Hormones / metabolism
  • Peptides / pharmacology
  • Polycystic Ovary Syndrome* / drug therapy
  • Polycystic Ovary Syndrome* / metabolism
  • Proto-Oncogene Proteins c-akt* / metabolism
  • Rats
  • Rats, Wistar*
  • Signal Transduction* / drug effects

Substances

  • NLR Family, Pyrin Domain-Containing 3 Protein
  • Glycogen Synthase Kinase 3 beta
  • NF-kappa B
  • Proto-Oncogene Proteins c-akt
  • ENHO protein, rat
  • Nlrp3 protein, rat
  • Intercellular Signaling Peptides and Proteins
  • Peptides
  • Peptide Hormones
  • Blood Proteins

Grants and funding

This research received no external funding from funding agencies in the public, commercial, or not-for-profit sectors.