Estrogen receptor-mediated cytotoxicity using iodine-125

J Cell Biochem. 1983;21(1):39-45. doi: 10.1002/jcb.240210106.

Abstract

Auger effects from 125I decay are singularly damaging if localized in DNA as the thymidine analogue 125I-iododeoxyuridine (125IUdR). Recent experience with steroid sex hormones extends these observations by demonstrating cytotoxicity in sites other than the DNA backbone. We have compared the cytotoxicity in human MCF-7 breast cancer cells of 125IUdR, 125I-iodotamoxifen, a nonsteroidal antiestrogen that is translocated from the cytoplasm to the nucleus of receptor containing cells, and 125I-iodoantipyrine, a biological indicator of the body water space. Cytotoxicity is critically dependent upon subcellular localization.

Publication types

  • Comparative Study
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Antipyrine / analogs & derivatives
  • Antipyrine / metabolism
  • Breast Neoplasms / metabolism
  • Breast Neoplasms / radiotherapy
  • Cell Line
  • Cell Survival / radiation effects*
  • Humans
  • Idoxuridine / metabolism
  • Iodine Radioisotopes / metabolism
  • Receptors, Estrogen / metabolism*
  • Tamoxifen / analogs & derivatives
  • Tamoxifen / metabolism

Substances

  • Iodine Radioisotopes
  • Receptors, Estrogen
  • Tamoxifen
  • iodotamoxifen
  • Idoxuridine
  • Antipyrine
  • iodoantipyrine