On the biosynthesis and metabolism of N-acylethanolamine phospholipids in infarcted dog heart

Biochim Biophys Acta. 1981 May 22;664(2):445-8. doi: 10.1016/0005-2760(81)90067-9.

Abstract

Minced tissues from both infarcted and apparently normal areas of canine myocardium 6 h after ligation of the left descending branch of the coronary artery were incubated with [1,2-(14)C]ethanolamine. In each case substantial amounts of radioactivity were incorporated into both phosphatidylethanolamine and N-acylphosphatidylethanolamine. Incubation of homogenates from the same tissues with N-[1-(14)C]palmitoyl phosphatidylethanolamine yielded labeled N-palmitoyl ethanolamine. The data support the concept that N-acylethanolamines, pharmacologically active compounds which accumulate in infarcted myocardium, are produced by N-acylation of ethanolamine phospholipids followed by phosphodiesterase action.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Acylation
  • Animals
  • Dogs
  • Ethanolamines / metabolism
  • Fatty Acids / metabolism
  • Ligation
  • Male
  • Metabolism*
  • Myocardial Infarction / metabolism*
  • Myocardium / metabolism*
  • Phosphatidylethanolamines / metabolism
  • Phospholipids

Substances

  • Ethanolamines
  • Fatty Acids
  • N-acylethanolamines
  • Phosphatidylethanolamines
  • Phospholipids