Abstract
NMDA and non-NMDA receptor-mediated components of excitatory post-synaptic current (EPSC) were studied by in situ whole-cell voltage-clamp recordings in the CA1 field of rat hippocampus. We found that the amplitudes ratio of the NMDA to the non-NMDA components can be strongly increased by blocking A1 adenosine receptors. The necessary conditions for this effect are both, increased Ca2+ and lowered Mg2+ in the external medium. The so achieved increase in the NMDA/non-NMDA ratio of EPSC components is irreversible and no longer depends on the activity of A1 adenosine receptors.
MeSH terms
-
Animals
-
Calcium / pharmacology*
-
Electrophysiology
-
Hippocampus / drug effects
-
Hippocampus / metabolism*
-
Hippocampus / physiology
-
In Vitro Techniques
-
Ion Channels / drug effects
-
Ion Channels / metabolism
-
Magnesium / pharmacology*
-
N-Methylaspartate / metabolism*
-
N-Methylaspartate / physiology
-
Neurons / drug effects
-
Neurons / physiology
-
Patch-Clamp Techniques
-
Purinergic P1 Receptor Antagonists*
-
Rats
-
Rats, Wistar
-
Receptors, Glutamate / drug effects
-
Receptors, Glutamate / metabolism*
-
Synapses / drug effects
-
Synapses / physiology*
-
Theophylline / analogs & derivatives
-
Theophylline / pharmacology
Substances
-
Ion Channels
-
Purinergic P1 Receptor Antagonists
-
Receptors, Glutamate
-
8-cyclopentyl-1,3-dimethylxanthine
-
N-Methylaspartate
-
Theophylline
-
Magnesium
-
Calcium