Interleukin-4 as a bone regulatory factor: effects on murine osteoblast-like cells

J Endocrinol Invest. 1995 Mar;18(3):174-9. doi: 10.1007/BF03347799.

Abstract

Bone remodelling is regulated at the local level by an incompletely elucidated cytokine network. In the present study we have determined the effect of interleukin-4 (IL-4), a cytokine produced by T lymphocytes and other cells, on the activity of murine osteoblasts. IL-4 (0.1-10 ng/ml) did not influence the proliferation rate of the osteoblast-like cell line MC3T3, but inhibited the expression of alkaline phosphatase. In long-term cultures supplemented with ascorbic acid and glycerophosphate such an effect was accompanied by a retardation of matrix mineralization. IL-4 also stimulated M-CSF expression by MC3T3 cells, both at the RNA and bioactivity levels. However, no stimulation of IL-1, IL-6, GM-CSF or PGE2 production was observed. An IL-4-induced inhibition of alkaline phosphatase expression and retardation of mineralization was also found in cultures of primary osteoblast-like cells isolated from neonatal mice calvariae. These results suggest that IL-4, probably released by cells within the bone marrow, may locally influence the activity of bone-forming cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Alkaline Phosphatase / metabolism
  • Animals
  • Ascorbic Acid / pharmacology
  • Bone Remodeling
  • Calcification, Physiologic
  • Cell Division
  • Cell Line
  • Interleukin-4 / pharmacology*
  • Macrophage Colony-Stimulating Factor / genetics
  • Macrophage Colony-Stimulating Factor / metabolism
  • Mice
  • Osteoblasts / cytology
  • Osteoblasts / metabolism*
  • Phosphatidylglycerols / pharmacology
  • RNA / metabolism

Substances

  • Phosphatidylglycerols
  • Interleukin-4
  • RNA
  • Macrophage Colony-Stimulating Factor
  • Alkaline Phosphatase
  • Ascorbic Acid