Activation of HIV expression by CD30 triggering in CD4+ T cells from HIV-infected individuals

Immunity. 1995 Aug;3(2):251-5. doi: 10.1016/1074-7613(95)90094-2.

Abstract

CD30 is a member of the tumor necrosis factor receptor superfamily, preferentially expressed by T cells producing type 2 helper (Th2) cytokines, whose ligand (CD30L) has been identified on B cells, activated macrophages, and a subset of activated T cells. We show here that cross-linking CD30 with an agonistic CD30-specific monoclonal antibody, as well as with CD30L+ CD8+ T cell clones or CD30L+ B cells, enhanced HIV replication in CD4+ T cells from HIV-infected individuals, and such a potentiating effect was inhibited by anti-CD30L antibody. The anti-CD30L antibody also exerted a suppressive effect on spontaneous HIV replication occurring in lymph node cells from an HIV-sero-positive patient, showing CD30L expression by both B and CD8+ T lymphocytes. Thus, CD30 triggering by CD30L-expressing cells may plan an important role in the activation of HIV expression from latently infected CD4+ T cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • B-Lymphocytes / physiology
  • CD3 Complex / physiology
  • CD30 Ligand
  • CD4-Positive T-Lymphocytes / microbiology*
  • CD8-Positive T-Lymphocytes / physiology
  • Cells, Cultured
  • HIV Infections / microbiology*
  • HIV-1 / growth & development*
  • Humans
  • Immunophenotyping
  • In Vitro Techniques
  • Ki-1 Antigen / physiology*
  • Membrane Glycoproteins / physiology*
  • Signal Transduction
  • Virus Replication*

Substances

  • CD3 Complex
  • CD30 Ligand
  • Ki-1 Antigen
  • Membrane Glycoproteins
  • TNFSF8 protein, human