Molecular recognition and assembly sequences involved in the subfamily-specific assembly of voltage-gated K+ channel subunit proteins

Neuron. 1995 Mar;14(3):625-33. doi: 10.1016/0896-6273(95)90319-4.

Abstract

We are analyzing features of the K+ channel subunit proteins that are critical for function and regulation of these proteins. Our studies show biochemically that subunit proteins from the Shaker and Shaw subfamilies fail to assemble into a heteromultimer. The basis for this incompatibility is the sequences contained within the T1 assembly domain. For a subunit protein to heteromultimerize with a Shaker subunit protein, two regions within the T1 domain, A and B, must be of the Shaker subtype. Finally, we show that the incompatibility of a Shaw A region for assembly with a Shaker protein depends upon the composition of a 30 amino acid conserved sequence in the A region.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Amino Acid Sequence
  • Animals
  • Aplysia
  • Cloning, Molecular
  • Conserved Sequence
  • Ion Channel Gating
  • Macromolecular Substances
  • Molecular Sequence Data
  • Mutagenesis, Site-Directed
  • Neurons / physiology*
  • Polymerase Chain Reaction
  • Potassium Channels / biosynthesis*
  • Potassium Channels / physiology
  • Protein Biosynthesis
  • Recombinant Proteins / biosynthesis
  • Sequence Homology, Amino Acid

Substances

  • Macromolecular Substances
  • Potassium Channels
  • Recombinant Proteins