Interleukin-4 inhibits the expression of tumour necrosis factors alpha and beta, interleukins-1 beta and -6 and interferon-gamma

Immunol Cell Biol. 1995 Feb;73(1):57-61. doi: 10.1038/icb.1995.9.

Abstract

T cell-derived IL-4 inhibits the expression of several inflammatory cytokine genes. In this study, IL-4 downregulated TNF-alpha and -beta, IL-1 beta IL-6 and IFN-gamma steady-state mRNA and protein production by blood leukocytes. In a detailed kinetic analysis, cytokine mRNA levels were significantly decreased in the presence of IL-4, suggesting an early and prolonged downregulatory effect. These findings support the general thesis that IL-4 is an important regulator of the inflammatory immune response.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Cytokines / biosynthesis*
  • Down-Regulation / immunology
  • Humans
  • Interferon-gamma / biosynthesis
  • Interleukin-1 / biosynthesis
  • Interleukin-4 / immunology*
  • Interleukin-6 / biosynthesis
  • Lymphotoxin-alpha / biosynthesis
  • RNA, Messenger / biosynthesis
  • Tumor Necrosis Factor-alpha / biosynthesis

Substances

  • Cytokines
  • Interleukin-1
  • Interleukin-6
  • Lymphotoxin-alpha
  • RNA, Messenger
  • Tumor Necrosis Factor-alpha
  • Interleukin-4
  • Interferon-gamma