Islet amyloidosis in a patient with chronic massive insulin resistance due to antiinsulin receptor antibodies

J Clin Endocrinol Metab. 1994 Jul;79(1):290-2. doi: 10.1210/jcem.79.1.8027243.

Abstract

We report islet hyperplasia, nesidioblastosis, and abundant islet amyloidosis (derived from islet amyloid polypeptide) in a case of severe insulin resistance due to a circulating antiinsulin receptor antibody. Massive chronic beta-cell stimulation (as well as noninsulin-dependent diabetes mellitus and insulinoma) appears to be associated with islet amyloid.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Amyloid / analysis
  • Amyloidosis / complications*
  • Amyloidosis / metabolism
  • Amyloidosis / pathology
  • Autoantibodies / blood*
  • Humans
  • Immunohistochemistry
  • Insulin Resistance / immunology*
  • Islet Amyloid Polypeptide
  • Islets of Langerhans* / chemistry
  • Islets of Langerhans* / pathology
  • Male
  • Middle Aged
  • Pancreatic Diseases / complications*
  • Pancreatic Diseases / metabolism
  • Pancreatic Diseases / pathology
  • Receptor, Insulin / immunology*
  • Ubiquitins / analysis

Substances

  • Amyloid
  • Autoantibodies
  • Islet Amyloid Polypeptide
  • Ubiquitins
  • Receptor, Insulin