Vasopressin antisense oligonucleotide induces temporary diabetes insipidus in rats

J Neuroendocrinol. 1994 Apr;6(2):121-5. doi: 10.1111/j.1365-2826.1994.tb00561.x.

Abstract

The purpose of this study was to downregulate the transcriptional message of arginine vasopressin (AVP) by antisense treatment. A complete phosphorothioate antisense oligodesoxynucleotide corresponding to the beginning of the coding region of rat AVP mRNA was constructed and injected into the lateral ventricle of rats. Within 3-6 h animals exhibited a temporary diabetes insipidus, which lasted up to 9 h. Accordingly, vasopressin immunoreactivity in the hypothalamic nuclei was reduced. Our results demonstrate that a specific and reversible inhibition of neuropeptide expression can be accomplished in the intact hypothalamo-neurohypophysial system by antisense treatment, thus providing a novel tool for studies on stimulus-secretion coupling in vivo.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Arginine Vasopressin / biosynthesis*
  • Base Sequence
  • Diabetes Insipidus / chemically induced*
  • Diabetes Insipidus / urine
  • Down-Regulation / drug effects
  • Injections, Intraventricular
  • Male
  • Molecular Sequence Data
  • Oligonucleotides, Antisense / administration & dosage
  • Oligonucleotides, Antisense / pharmacology*
  • Osmolar Concentration
  • Rats
  • Rats, Wistar

Substances

  • Oligonucleotides, Antisense
  • Arginine Vasopressin