Effect of intracellular Ca2+ concentration on endothelin-1 secretion

FEBS Lett. 1994 Aug 15;350(1):33-6. doi: 10.1016/0014-5793(94)00727-6.

Abstract

The role of intracellular free Ca2+ concentration ([Ca2+]i) in cellular regulation of endothelin-1 (ET-1) secretion was investigated in cultured porcine aortic endothelial cells of first passage. Intracellular Ca2+ concentrations were adjusted between 50 nM and 1 microM using EGTA and thapsigargin, respectively. ET-1 secretion was maximal at [Ca2+]i of 190-470 nM, and reduced at low (50 and 110 nM) and high (> 470 nM) [Ca2+]i. The Ca2+ ionophores A23187 and ionomycin (each 1 microM), both of which raise [Ca2+]i above 1 microM, also potently inhibited ET-1 secretion under basal and stimulated conditions. The A23187-induced reduction in ET-1 secretion was not affected by NG-nitro-L-arginine (0.1 mM). Our results provide evidence that basal ET-1 secretion is regulated by Ca2+ and that Ca2+ ionophores reduce ET-1 secretion due to the inhibitory effect of high [Ca2+]i.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Calcimycin / pharmacology
  • Calcium / metabolism*
  • Cells, Cultured
  • Endothelins / metabolism*
  • Endothelium, Vascular / drug effects
  • Endothelium, Vascular / enzymology
  • Endothelium, Vascular / metabolism*
  • Ionomycin / pharmacology
  • L-Lactate Dehydrogenase / metabolism
  • Swine
  • Tetradecanoylphorbol Acetate / pharmacology

Substances

  • Endothelins
  • Calcimycin
  • Ionomycin
  • L-Lactate Dehydrogenase
  • Tetradecanoylphorbol Acetate
  • Calcium