Staphylococcal enterotoxin B promotes deletion and functional inactivation of CD4V beta 8-positive cells in the absence of CD8 T cells

Scand J Immunol. 1993 Sep;38(3):254-8. doi: 10.1111/j.1365-3083.1993.tb01722.x.

Abstract

The staphylococcal enterotoxins stimulate discrete subsets of T cells depending on their expression of particular V genes. Among these, staphylococcal enterotoxin B (SEB) vigorously stimulates V beta 8+ cells. This stimulation results in proliferation of both CD4+V beta 8+ and CD8+ T cells and eventually to anergy and clonal deletion in the former subset. We have examined the possible role of CD8+ T cells in the response of CD4+ cells to SEB, by in vivo CD8+ T-cell-depletion. We found no qualitative difference in the responses of untreated and CD8+ T-cell depleted mice to SEB; however, a small quantitative difference in deletion was observed. Thus it appears that on the whole the response of CD4+V beta 8+ T cells to SEB is independent of CD8+ T-cell effector function, although the latter may play a partial role.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • CD4-Positive T-Lymphocytes / drug effects*
  • CD4-Positive T-Lymphocytes / physiology
  • Enterotoxins / pharmacology*
  • Flow Cytometry
  • Lymphocyte Activation / drug effects
  • Mice
  • Mice, Inbred BALB C
  • Receptors, Antigen, T-Cell / analysis
  • Receptors, Antigen, T-Cell, alpha-beta*
  • Spleen / cytology
  • Staphylococcus aureus
  • T-Lymphocytes, Cytotoxic / physiology*
  • T-Lymphocytes, Regulatory / physiology*

Substances

  • Enterotoxins
  • Receptors, Antigen, T-Cell
  • Receptors, Antigen, T-Cell, alpha-beta
  • T cell receptor Vbeta8
  • enterotoxin B, staphylococcal