Correlation of soluble interleukin-2 receptor levels with severity of chronic hepatitis C virus liver injury and development of hepatocellular cancer

Surgery. 1996 Jul;120(1):100-5. doi: 10.1016/s0039-6060(96)80247-9.

Abstract

Background: Patients with acute and chronic hepatitis B virus infection have elevated serum levels of soluble interleukin-2 receptor. This study examined patients with chronic hepatitis C virus infection to determine whether serum soluble interleukin-2 receptor levels were elevated and whether the degree of these elevations in serum levels correlated with histologic severity of hepatitis-related liver injury.

Methods: Percutaneous liver biopsies were performed on 123 patients with chronic hepatitis C virus infection. Serum levels of soluble interleukin-2 receptor in these 123 patients were measured by means of specific enzyme-linked immunoassay and were compared with levels in 174 subjects in a hepatitis-free control group.

Results: Soluble interleukin-2 receptor levels were significantly higher in the patients with hepatitis C than in subjects in a control group (p < 0.0001). A progressive and significant increase occurred in soluble interleukin-2 receptor levels with increasing severity of liver injury (p < 0.001). The highest levels of soluble interleukin-2 receptor occurred in patients who had hepatocellular cancer.

Conclusions: Soluble interleukin-2 receptor levels correlate with the histologic severity of liver damage in patients with chronic hepatitis C virus infection and may be useful as a marker in patients at high risk of getting hepatocellular cancer.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Aged
  • Carcinoma, Hepatocellular / etiology*
  • Chronic Disease
  • Female
  • Hepatitis C / blood
  • Hepatitis C / complications
  • Hepatitis C / pathology*
  • Humans
  • Liver / pathology*
  • Liver Neoplasms / etiology*
  • Male
  • Middle Aged
  • Receptors, Interleukin-2 / analysis*
  • alpha-Fetoproteins / analysis

Substances

  • Receptors, Interleukin-2
  • alpha-Fetoproteins