An Iranian-Armenian LDLR frameshift mutation causing familial hypercholesterolemia

Clin Genet. 1996 Feb;49(2):88-90. doi: 10.1111/j.1399-0004.1996.tb04334.x.

Abstract

We used polymerase chain reaction single-strand conformation polymorphism (PCR-SSCP) analysis to detect a mutation in the low density lipoprotein receptor (LDLR) gene in a family of Iranian-Armenian origin. The mutation, designated FH Yrmeih, deletes two nucleotides from exon 10 of the LDLR gene, which causes a translational frameshift, whereby a truncated LDLR protein of the first 471 residues of the LDLR with an additional 41 abnormal residues and a premature stop codon would be created. The deletion was detected in a father and son with clinical features of heterozygous FH. To our knowledge this is the first pathogenetic LDLR mutation identified in FH patients of Iranian-Armenian ancestry.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Armenia / ethnology
  • Base Sequence
  • Cholesterol / analysis
  • Cholesterol, HDL / analysis
  • Cholesterol, LDL / analysis
  • Denmark
  • Frameshift Mutation*
  • Humans
  • Hyperlipoproteinemia Type II / ethnology
  • Hyperlipoproteinemia Type II / genetics*
  • Iran / ethnology
  • Male
  • Middle Aged
  • Molecular Sequence Data
  • Polymerase Chain Reaction
  • Polymorphism, Single-Stranded Conformational
  • Receptors, LDL / genetics*
  • Triglycerides / analysis

Substances

  • Cholesterol, HDL
  • Cholesterol, LDL
  • Receptors, LDL
  • Triglycerides
  • Cholesterol