Inhibition of nitric oxide formation reduces voluntary ethanol consumption in the rat

Psychopharmacology (Berl). 1996 Jun;125(4):398-401. doi: 10.1007/BF02246024.

Abstract

Brain nitric oxide is involved in the mechanisms that regulate ingestive behavior. To test whether this compound plays a role in alcohol preference, we studied the effects of different doses of NG-nitro-L-arginine (L-NO arg), an inhibitor of nitric oxide synthase (NOS), on voluntary consumption of ethanol and on blood alcohol levels produced by a single intraperitoneal dose of alcohol in the rat. L-NO arg produced a significant and dose-dependent reduction of ethanol intake (P < 0.001) without influencing total fluid consumption or feeding behavior. L-NO arg did not influence the kinetics of alcohol. Our data show that inhibition of nitric oxide formation accompanies reduction of ethanol intake and suggest a possible role for nitric oxide in ethanol self-administration.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Alcohol Drinking / physiopathology*
  • Animals
  • Dose-Response Relationship, Drug
  • Enzyme Inhibitors / pharmacology*
  • Ethanol / blood*
  • Male
  • Nitric Oxide Synthase / antagonists & inhibitors*
  • Nitroarginine / pharmacology*
  • Rats
  • Rats, Sprague-Dawley
  • Self Administration

Substances

  • Enzyme Inhibitors
  • Nitroarginine
  • Ethanol
  • Nitric Oxide Synthase