Human immunodeficiency virus reverse transcriptase codon 215 mutations diminish virologic response to didanosine-zidovudine therapy in subjects with non-syncytium-inducing phenotype

J Infect Dis. 1996 Oct;174(4):854-7. doi: 10.1093/infdis/174.4.854.

Abstract

Eight zidovudine-experienced subjects received zidovudine and didanosine for 30 weeks followed by 30 weeks of didanosine monotherapy. At study entry, plasma from 4 subjects had human immunodeficiency virus RNA pol T215Y/F mutant and 4 had codon 215 wild type. All 8 subjects had non-syncytium-inducing virus phenotype. Sustained 10-fold decreases in plasma RNA levels were seen only in subjects who initially had 215 wild type RNA, despite the development of a T215Y/F mutation during combination therapy. Virologic and immunologic benefits were maintained in this group with didanosine monotherapy. No subject developed a pol L74V codon mutation. Significant differences in plasma virus load and CD4 cell responses were seen in this zidovudine-didanosine combination pilot study relative to codon 215 genotype.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acquired Immunodeficiency Syndrome / drug therapy*
  • Adult
  • Anti-HIV Agents / administration & dosage*
  • CD4 Lymphocyte Count
  • Codon*
  • Didanosine / administration & dosage*
  • Drug Therapy, Combination
  • HIV Reverse Transcriptase / genetics*
  • Humans
  • Male
  • Middle Aged
  • Mutation*
  • Phenotype
  • RNA, Viral / blood
  • Zidovudine / administration & dosage*

Substances

  • Anti-HIV Agents
  • Codon
  • RNA, Viral
  • Zidovudine
  • HIV Reverse Transcriptase
  • Didanosine