Autoimmunity due to defective Nur77, Fas, and TNF-RI apoptosis

Adv Exp Med Biol. 1996:406:241-62. doi: 10.1007/978-1-4899-0274-0_25.
No abstract available

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.
  • Review

MeSH terms

  • Animals
  • Antigens, CD / genetics
  • Antigens, CD / immunology*
  • Apoptosis / immunology
  • Autoimmunity / immunology*
  • DNA-Binding Proteins / genetics
  • DNA-Binding Proteins / immunology*
  • Gene Deletion
  • Humans
  • Inflammation
  • Macrophages / physiology
  • Mice
  • Mice, Inbred MRL lpr
  • Mice, Transgenic
  • Nuclear Receptor Subfamily 4, Group A, Member 1
  • Receptors, Cytoplasmic and Nuclear
  • Receptors, Steroid
  • Receptors, Tumor Necrosis Factor / genetics
  • Receptors, Tumor Necrosis Factor / immunology*
  • Receptors, Tumor Necrosis Factor, Type I
  • Shock, Septic
  • Signal Transduction / immunology*
  • T-Lymphocytes / immunology
  • Transcription Factors / genetics
  • Transcription Factors / immunology*
  • fas Receptor / genetics
  • fas Receptor / immunology*

Substances

  • Antigens, CD
  • DNA-Binding Proteins
  • NR4A1 protein, human
  • Nr4a1 protein, mouse
  • Nuclear Receptor Subfamily 4, Group A, Member 1
  • Receptors, Cytoplasmic and Nuclear
  • Receptors, Steroid
  • Receptors, Tumor Necrosis Factor
  • Receptors, Tumor Necrosis Factor, Type I
  • Transcription Factors
  • fas Receptor