Mutation rates in LTR of HTLV-1 in HAM/TSP patients and the carriers are similarly high to Tax/Rex-coding sequence

J Neurovirol. 1996 Oct;2(5):330-5. doi: 10.3109/13550289609146897.

Abstract

The genomic sequence of human T-cell leukemia virus type 1 (HTLV-1) is highly conserved, although minor sequence variations enable classification of the isolates into several subgroups. We previously reported, however, that the Tax-coding sequence of HTLV-1 genome is highly variable in a random fashion within individuals with HAM/TSP and asymptomatic carriers. Here, we describe frequent base substitutions in the LTR sequence similarly to those in Tax-coding sequence. These observations indicate that frequent mutations are not unique to the sequence encoding the most effective antigen for cytotoxic T lymphocytes, but also seen in the LTR, a non-coding sequence. Thus, frequent mutations seem to occur during the viral replication process rather than the selection of rare mutants by immune surveillance.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Base Sequence
  • Carrier State / virology*
  • DNA Mutational Analysis
  • DNA, Viral / analysis
  • DNA, Viral / genetics
  • Genetic Variation
  • Human T-lymphotropic virus 1 / genetics*
  • Humans
  • Mutation / genetics
  • Paraparesis, Tropical Spastic / virology*
  • Polymerase Chain Reaction
  • Repetitive Sequences, Nucleic Acid*

Substances

  • DNA, Viral