Identification of Ser-1275 and Ser-1309 as autophosphorylation sites of the insulin receptor

FEBS Lett. 1997 Jan 2;400(1):65-70. doi: 10.1016/s0014-5793(96)01342-7.

Abstract

We have identified Ser-1275 and Ser-1309 as novel serine autophosphorylation sites by direct sequencing of HPLC-purified tryptic phosphopeptides of the histidine-tagged insulin receptor kinase IRKD-HIS. The corresponding peptides (Ser-1275, amino acids 1272-1292; Ser-1309, amino acids 1305-1313) have been detected in the HPLC profiles of both the soluble kinase IRKD, which contains the entire cytoplasmic domain of the insulin receptor beta-subunit, and the insulin receptor purified from human placenta. In contrast, a kinase negative mutant, IRKD-K1018A, did not undergo phosphorylation at either the tyrosine or serine residues, strongly suggesting that insulin receptor kinase has an intrinsic activity to autophosphorylate serine residues.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Animals
  • Cell Line
  • Humans
  • Molecular Sequence Data
  • Mutation
  • Peptide Mapping
  • Phosphopeptides / metabolism
  • Phosphorylation
  • Receptor, Insulin / genetics
  • Receptor, Insulin / isolation & purification
  • Receptor, Insulin / metabolism*
  • Recombinant Proteins / metabolism
  • Serine / metabolism*
  • Solubility
  • Spodoptera
  • Threonine / metabolism

Substances

  • Phosphopeptides
  • Recombinant Proteins
  • Threonine
  • Serine
  • Receptor, Insulin