Circadian aqueous flow mediated by beta-arrestin induced homologous desensitization

Exp Eye Res. 1997 Jun;64(6):1005-11. doi: 10.1006/exer.1997.0298.

Abstract

It was hypothesized that homologous desensitization regulates signal transduction from the beta-adrenergic receptor in the ocular ciliary epithelium to affect the circadian rhythm of aqueous humor secretion. beta-arrestin-1 was cloned from the rabbit ciliary epithelium, and the full length cDNA used as a probe for Northern blot analysis to examine the diurnal expression of beta-arrestin mRNA. Protein expression of beta-arrestin-1 at intervals during the circadian cycle of aqueous secretion showed a decrease in beta-arrestin expression when maximal activation of the beta-adrenergic receptor is known to increase secretion. Diurnal expression of beta-arrestin suggests that homologous desensitization can regulate the circadian rhythm of aqueous flow.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Amino Acid Sequence
  • Animals
  • Aqueous Humor / physiology*
  • Arrestin / genetics
  • Arrestin / metabolism*
  • Arrestin / physiology
  • Blotting, Northern
  • Blotting, Western
  • Ciliary Body / metabolism
  • Circadian Rhythm / physiology*
  • Epithelium / metabolism
  • Fluorescent Antibody Technique, Indirect
  • Gene Expression Regulation
  • Male
  • Molecular Sequence Data
  • RNA, Messenger / genetics
  • Rabbits
  • Species Specificity

Substances

  • Arrestin
  • RNA, Messenger

Associated data

  • GENBANK/U75838