L-selectin activates JNK via src-like tyrosine kinases and the small G-protein Rac

Immunology. 1997 Oct;92(2):214-9. doi: 10.1046/j.1365-2567.1997.00336.x.

Abstract

Selectin and alpha 4 beta 7-integrins have been shown to mediate transient leucocyte interactions with endothelial cells which is a crucial step in the initial immune response to pathogens. We have previously shown that stimulation of T lymphocytes via L-selectin results in activation of a signalling cascade from the L-selectin molecule via the tyrosine kinase p56lck and tyrosine phosphorylation of L-selectin to the stimulation of p21Ras and Rac proteins. In the present study we demonstrate that stimulation of Jurkat T lymphocytes via L-selectin results in an activation of Jun N-terminal kinase (JNK) but not of p38-K. L-selectin-initiated activation of JNK is mediated by src-like tyrosine kinases and the small G-protein Rac 1/2, since genetic or pharmacological inhibition of p56lck or Rac proteins prevent the stimulation of JNK by L-selectin. Thus, the data point to a novel signalling cascade from L-selectin via src-like tyrosine kinases and Rac proteins to JNK.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Blotting, Western
  • Calcium-Calmodulin-Dependent Protein Kinases / metabolism*
  • Electrophoresis, Polyacrylamide Gel
  • GTP-Binding Proteins / metabolism*
  • Humans
  • JNK Mitogen-Activated Protein Kinases
  • L-Selectin / pharmacology*
  • Lymphocyte Activation / physiology
  • Lymphocyte Specific Protein Tyrosine Kinase p56(lck) / metabolism*
  • Mitogen-Activated Protein Kinases*
  • Signal Transduction / physiology
  • T-Lymphocytes / drug effects*
  • T-Lymphocytes / metabolism
  • Tumor Cells, Cultured
  • rac GTP-Binding Proteins

Substances

  • L-Selectin
  • Lymphocyte Specific Protein Tyrosine Kinase p56(lck)
  • Calcium-Calmodulin-Dependent Protein Kinases
  • JNK Mitogen-Activated Protein Kinases
  • Mitogen-Activated Protein Kinases
  • GTP-Binding Proteins
  • rac GTP-Binding Proteins