Deriving components of genetic variance for multilocus models

Genet Epidemiol. 1997;14(6):1131-6. doi: 10.1002/(SICI)1098-2272(1997)14:6<1131::AID-GEPI95>3.0.CO;2-H.

Abstract

Several authors have considered two-locus models as a basis for the inheritance of complex diseases. The purpose of this paper is to give a simple general formulation to derive the additive, dominant, and epistatic effects, and hence the corresponding variance components, for any multilocus model. These variance components should be useful for investigating the power of model-free linkage analysis to detect various modes of multilocus inheritance.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Alleles
  • Chromosome Mapping
  • Epistasis, Genetic
  • Female
  • Gene Frequency*
  • Genetic Diseases, Inborn / genetics*
  • Genetic Linkage*
  • Genetic Variation*
  • Humans
  • Male
  • Matched-Pair Analysis
  • Models, Genetic*
  • Nuclear Family
  • Penetrance