Abstract
Shiga toxin-producing Escherichia coli O111:H2 strains from an outbreak of hemolytic-uremic syndrome showed aggregative adhesion to HEp-2 cells and harbored large plasmids which hybridized with the enteroaggregative E. coli probe PCVD432. These strains present a novel combination of virulence factors and might be as pathogenic to humans as the classic enterohemorrhagic E. coli.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Bacterial Adhesion*
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Bacterial Toxins / biosynthesis*
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Coliphages / genetics
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Coliphages / physiology
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Disease Outbreaks*
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Escherichia coli / genetics
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Escherichia coli / pathogenicity*
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Escherichia coli Infections / epidemiology
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Escherichia coli Infections / microbiology*
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Hemolytic-Uremic Syndrome / epidemiology
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Hemolytic-Uremic Syndrome / microbiology*
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Humans
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Plasmids
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Shiga Toxins
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Tumor Cells, Cultured
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Virulence
Substances
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Bacterial Toxins
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Shiga Toxins