Inhibition of establishment and growth of mouse liver metastases after treatment with interferon gamma and beta-1,3-D-glucan

Hepatology. 1998 May;27(5):1241-8. doi: 10.1002/hep.510270509.

Abstract

The purpose of this study was to investigate the combined antitumor effect of aminated beta-1,3-D-glucan (AG) and interferon-gamma (IFN-gamma) in an experimental liver metastasis model. Liver metastases were established by inoculation of C-26 colon carcinoma cells into the superior mesenteric vein of syngeneic mice. Treatment of mice started 24 hours after inoculation of tumor cells by daily intravenous injections of either AG, IFN-gamma, or a combination of both for a duration of 6 days. The resultant liver metastases were then quantified after an additional period of 11 days. Combination of IFN-gamma and AG inhibited the growth of liver metastases almost entirely. IFN-gamma was also very efficient, while AG alone did not exert any significant antitumor effect. These results, along with histological studies from mice receiving AG and IFN-gamma, indicated that activation and recruitment of liver macrophages may be a part of the mechanism responsible for the inhibition of metastatic growth observed in this study.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antineoplastic Agents / administration & dosage*
  • Carcinoma / pathology*
  • Colonic Neoplasms / pathology*
  • Drug Synergism
  • Female
  • Glucans / pharmacology*
  • Interferon-gamma / pharmacology*
  • Interferon-gamma / therapeutic use
  • Liver Neoplasms / immunology
  • Liver Neoplasms / pathology
  • Liver Neoplasms / secondary*
  • Mice
  • Mice, Inbred BALB C
  • Mice, Inbred C57BL
  • Neoplasm Metastasis / prevention & control*
  • Recombinant Proteins
  • beta-Glucans*

Substances

  • Antineoplastic Agents
  • Glucans
  • Recombinant Proteins
  • beta-Glucans
  • Interferon-gamma
  • beta-1,3-glucan