Abstract
1. BAPTA AM (10 microM), thapsigargin (10 microM), ruthenium red (30 microM) and oligomycin (30 microM) inhibited field-stimulated [3H]NE release from bovine isolated irides by 54%, 30%, 30% and 26%, respectively. 2. Both BAPTA AM and thapsigargin had no significant effect (P>0.05) on H2O2-induced potentiation of evoked [3H]NE release. 3. Ruthenium red prevented (but oligomycin enhanced) H2O2-induced enhancement of evoked [3H]NE release. 4. We conclude that, although intracellular calcium participates in field-stimulation evoked [3H]NE release from bovine isolated irides, only the mitochondrial pool of calcium may be involved in peroxide-induced enhancement of sympathetic neurotransmission.
Publication types
-
Research Support, U.S. Gov't, P.H.S.
MeSH terms
-
Animals
-
Calcium / antagonists & inhibitors
-
Calcium / metabolism*
-
Calcium-Transporting ATPases / antagonists & inhibitors
-
Cattle
-
Chelating Agents / pharmacology
-
Drug Synergism
-
Egtazic Acid / analogs & derivatives
-
Egtazic Acid / pharmacology
-
Electric Stimulation
-
Enzyme Inhibitors / pharmacology
-
Homeostasis
-
Hydrogen Peroxide / pharmacology*
-
In Vitro Techniques
-
Intracellular Fluid / drug effects
-
Intracellular Fluid / metabolism*
-
Iris / drug effects*
-
Iris / metabolism
-
Iris / ultrastructure
-
Mitochondria / drug effects
-
Mitochondria / metabolism
-
Norepinephrine / metabolism*
-
Oligomycins / pharmacology
-
Ruthenium Red / pharmacology
-
Thapsigargin / pharmacology
Substances
-
Chelating Agents
-
Enzyme Inhibitors
-
Oligomycins
-
Ruthenium Red
-
Egtazic Acid
-
Thapsigargin
-
Hydrogen Peroxide
-
Calcium-Transporting ATPases
-
1,2-bis(2-aminophenoxy)ethane-N,N,N',N'-tetraacetic acid
-
Calcium
-
Norepinephrine