Detection of anti-colon antibodies in inflammatory bowel disease using human cultured colonic cells

Gut. 1999 Feb;44(2):196-202. doi: 10.1136/gut.44.2.196.

Abstract

Background: Investigation of anti-colon antibodies may be simplified if a sensitive method and homogeneous source of antigen were available.

Aims: To examine the anti-colon antibody response using human colonic carcinoma cell lines as antigen.

Subjects: Patients with inflammatory bowel disease and other gastrointestinal disorders and healthy controls were studied.

Methods: Comparative enzyme linked immunosorbent assays (ELISAs) were performed to assess the value of whole Caco-2, HT-29, and LS-180 cells as antigen. The antigenic determinants of the immune response were characterised by western blot analysis.

Results: Sera demonstrated immunoreactivity against each of the cell lines, but different epitopes were recognised. Applying whole Caco-2 cells as antigen in an ELISA, the prevalence of anti-colon antibodies was significantly greater in patients with ulcerative colitis (36%) than Crohn's disease (13%), other gastrointestinal disorders (13%) and healthy controls (0) (p<0. 05). The immune response was not associated with one predominant antigen.

Conclusions: Fixed whole cell ELISA is a simple and feasible method for studying the anti-colon antibody response. This response is non-specific, being directed against multiple antigens, and is likely to be an epiphenomenon of inflammatory bowel disease, more so for ulcerative colitis than Crohn's disease.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Aged
  • Autoantibodies / biosynthesis*
  • Autoantibodies / blood
  • Autoantigens / immunology
  • Blotting, Western
  • Cell Culture Techniques
  • Cell Line
  • Colitis, Ulcerative / immunology*
  • Colon / immunology*
  • Crohn Disease / immunology*
  • Electrophoresis, Polyacrylamide Gel
  • Enzyme-Linked Immunosorbent Assay
  • Epitopes / blood
  • Humans
  • Middle Aged

Substances

  • Autoantibodies
  • Autoantigens
  • Epitopes