Amalfitano A - Search Results

1

Persistence of an [E1-, polymerase-] adenovirus vector despite transduction of a neoantigen into immune-competent mice.

Hu H, Serra D, Amalfitano A. Hu H, et al. Among authors: amalfitano a. Hum Gene Ther. 1999 Feb 10;10(3):355-64. doi: 10.1089/10430349950018805. Hum Gene Ther. 1999. PMID: 10048388

2

Production and characterization of improved adenovirus vectors with the E1, E2b, and E3 genes deleted.

Amalfitano A, Hauser MA, Hu H, Serra D, Begy CR, Chamberlain JS. Amalfitano A, et al. J Virol. 1998 Feb;72(2):926-33. doi: 10.1128/JVI.72.2.926-933.1998. J Virol. 1998. PMID: 9444984 Free PMC article.

3

Multiply deleted [E1, polymerase-, and pTP-] adenovirus vector persists despite deletion of the preterminal protein.

Hodges BL, Serra D, Hu H, Begy CA, Chamberlain JS, Amalfitano A. Hodges BL, et al. Among authors: amalfitano a. J Gene Med. 2000 Jul-Aug;2(4):250-9. doi: 10.1002/1521-2254(200007/08)2:4<250::AID-JGM113>3.0.CO;2-3. J Gene Med. 2000. PMID: 10953916

4

Long-term efficacy after [E1-, polymerase-] adenovirus-mediated transfer of human acid-alpha-glucosidase gene into glycogen storage disease type II knockout mice.

Ding EY, Hodges BL, Hu H, McVie-Wylie AJ, Serra D, Migone FK, Pressley D, Chen YT, Amalfitano A. Ding EY, et al. Among authors: amalfitano a. Hum Gene Ther. 2001 May 20;12(8):955-65. doi: 10.1089/104303401750195917. Hum Gene Ther. 2001. PMID: 11387060

5

Adenovirus vectors with the 100K gene deleted and their potential for multiple gene therapy applications.

Hodges BL, Evans HK, Everett RS, Ding EY, Serra D, Amalfitano A. Hodges BL, et al. Among authors: amalfitano a. J Virol. 2001 Jul;75(13):5913-20. doi: 10.1128/JVI.75.13.5913-5920.2001. J Virol. 2001. PMID: 11390592 Free PMC article.

6

Multiple muscles in the AMD quail can be "cross-corrected" of pathologic glycogen accumulation after intravenous injection of an [E1-, polymerase-] adenovirus vector encoding human acid-alpha-glucosidase.

McVie-Wylie AJ, Ding EY, Lawson T, Serra D, Migone FK, Pressley D, Mizutani M, Kikuchi T, Chen YT, Amalfitano A. McVie-Wylie AJ, et al. Among authors: amalfitano a. J Gene Med. 2003 May;5(5):399-406. doi: 10.1002/jgm.355. J Gene Med. 2003. PMID: 12731088

7

Liver toxicities typically induced by first-generation adenoviral vectors can be reduced by use of E1, E2b-deleted adenoviral vectors.

Everett RS, Hodges BL, Ding EY, Xu F, Serra D, Amalfitano A. Everett RS, et al. Among authors: amalfitano a. Hum Gene Ther. 2003 Dec 10;14(18):1715-26. doi: 10.1089/104303403322611737. Hum Gene Ther. 2003. PMID: 14670123

8

Improved efficacy of gene therapy approaches for Pompe disease using a new, immune-deficient GSD-II mouse model.

Xu F, Ding E, Liao SX, Migone F, Dai J, Schneider A, Serra D, Chen YT, Amalfitano A. Xu F, et al. Among authors: amalfitano a. Gene Ther. 2004 Nov;11(21):1590-8. doi: 10.1038/sj.gt.3302314. Gene Ther. 2004. PMID: 15356673

9

Recombinant adenovirus vectors activate the alternative complement pathway, leading to the binding of human complement protein C3 independent of anti-ad antibodies.

Jiang H, Wang Z, Serra D, Frank MM, Amalfitano A. Jiang H, et al. Among authors: amalfitano a. Mol Ther. 2004 Dec;10(6):1140-2. doi: 10.1016/j.ymthe.2004.08.015. Mol Ther. 2004. PMID: 15564145 Free article.

10

Systemic correction of the muscle disorder glycogen storage disease type II after hepatic targeting of a modified adenovirus vector encoding human acid-alpha-glucosidase.

Amalfitano A, McVie-Wylie AJ, Hu H, Dawson TL, Raben N, Plotz P, Chen YT. Amalfitano A, et al. Proc Natl Acad Sci U S A. 1999 Aug 3;96(16):8861-6. doi: 10.1073/pnas.96.16.8861. Proc Natl Acad Sci U S A. 1999. PMID: 10430861 Free PMC article.

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