Tschopp J - Search Results

1

Fas ligand-induced c-Jun kinase activation in lymphoid cells requires extensive receptor aggregation but is independent of DAXX, and Fas-mediated cell death does not involve DAXX, RIP, or RAIDD.

Villunger A, Huang DC, Holler N, Tschopp J, Strasser A. Villunger A, et al. Among authors: tschopp j. J Immunol. 2000 Aug 1;165(3):1337-43. doi: 10.4049/jimmunol.165.3.1337. J Immunol. 2000. PMID: 10903735

2

Activation of Fas by FasL induces apoptosis by a mechanism that cannot be blocked by Bcl-2 or Bcl-x(L).

Huang DC, Hahne M, Schroeter M, Frei K, Fontana A, Villunger A, Newton K, Tschopp J, Strasser A. Huang DC, et al. Among authors: tschopp j. Proc Natl Acad Sci U S A. 1999 Dec 21;96(26):14871-6. doi: 10.1073/pnas.96.26.14871. Proc Natl Acad Sci U S A. 1999. PMID: 10611305 Free PMC article.

3

Activation of a pro-apoptotic amplification loop through inhibition of NF-kappaB-dependent survival signals by caspase-mediated inactivation of RIP.

Martinon F, Holler N, Richard C, Tschopp J. Martinon F, et al. Among authors: tschopp j. FEBS Lett. 2000 Feb 25;468(2-3):134-6. doi: 10.1016/s0014-5793(00)01212-6. FEBS Lett. 2000. PMID: 10692573 Free article.

4

Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule.

Holler N, Zaru R, Micheau O, Thome M, Attinger A, Valitutti S, Bodmer JL, Schneider P, Seed B, Tschopp J. Holler N, et al. Among authors: tschopp j. Nat Immunol. 2000 Dec;1(6):489-95. doi: 10.1038/82732. Nat Immunol. 2000. PMID: 11101870

5

The long form of FLIP is an activator of caspase-8 at the Fas death-inducing signaling complex.

Micheau O, Thome M, Schneider P, Holler N, Tschopp J, Nicholson DW, Briand C, Grütter MG. Micheau O, et al. Among authors: tschopp j. J Biol Chem. 2002 Nov 22;277(47):45162-71. doi: 10.1074/jbc.M206882200. Epub 2002 Sep 4. J Biol Chem. 2002. PMID: 12215447 Free article.

6

Two adjacent trimeric Fas ligands are required for Fas signaling and formation of a death-inducing signaling complex.

Holler N, Tardivel A, Kovacsovics-Bankowski M, Hertig S, Gaide O, Martinon F, Tinel A, Deperthes D, Calderara S, Schulthess T, Engel J, Schneider P, Tschopp J. Holler N, et al. Among authors: tschopp j. Mol Cell Biol. 2003 Feb;23(4):1428-40. doi: 10.1128/MCB.23.4.1428-1440.2003. Mol Cell Biol. 2003. PMID: 12556501 Free PMC article.

7

Characterization of Fas (Apo-1, CD95)-Fas ligand interaction.

Schneider P, Bodmer JL, Holler N, Mattmann C, Scuderi P, Terskikh A, Peitsch MC, Tschopp J. Schneider P, et al. Among authors: tschopp j. J Biol Chem. 1997 Jul 25;272(30):18827-33. doi: 10.1074/jbc.272.30.18827. J Biol Chem. 1997. PMID: 9228058 Free article.

8

Characterization of two receptors for TRAIL.

Schneider P, Bodmer JL, Thome M, Hofmann K, Holler N, Tschopp J. Schneider P, et al. Among authors: tschopp j. FEBS Lett. 1997 Oct 27;416(3):329-34. doi: 10.1016/s0014-5793(97)01231-3. FEBS Lett. 1997. PMID: 9373179 Free article.

9

TRAIL receptors 1 (DR4) and 2 (DR5) signal FADD-dependent apoptosis and activate NF-kappaB.

Schneider P, Thome M, Burns K, Bodmer JL, Hofmann K, Kataoka T, Holler N, Tschopp J. Schneider P, et al. Among authors: tschopp j. Immunity. 1997 Dec;7(6):831-6. doi: 10.1016/s1074-7613(00)80401-x. Immunity. 1997. PMID: 9430228 Free article.

10

Conversion of membrane-bound Fas(CD95) ligand to its soluble form is associated with downregulation of its proapoptotic activity and loss of liver toxicity.

Schneider P, Holler N, Bodmer JL, Hahne M, Frei K, Fontana A, Tschopp J. Schneider P, et al. Among authors: tschopp j. J Exp Med. 1998 Apr 20;187(8):1205-13. doi: 10.1084/jem.187.8.1205. J Exp Med. 1998. PMID: 9547332 Free PMC article.

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