Nip J - Search Results

1

TEL, a putative tumor suppressor, modulates cell growth and cell morphology of ras-transformed cells while repressing the transcription of stromelysin-1.

Fenrick R, Wang L, Nip J, Amann JM, Rooney RJ, Walker-Daniels J, Crawford HC, Hulboy DL, Kinch MS, Matrisian LM, Hiebert SW. Fenrick R, et al. Among authors: nip j. Mol Cell Biol. 2000 Aug;20(16):5828-39. doi: 10.1128/MCB.20.16.5828-5839.2000. Mol Cell Biol. 2000. PMID: 10913166 Free PMC article.

2

ETO, a target of t(8;21) in acute leukemia, makes distinct contacts with multiple histone deacetylases and binds mSin3A through its oligomerization domain.

Amann JM, Nip J, Strom DK, Lutterbach B, Harada H, Lenny N, Downing JR, Meyers S, Hiebert SW. Amann JM, et al. Among authors: nip j. Mol Cell Biol. 2001 Oct;21(19):6470-83. doi: 10.1128/MCB.21.19.6470-6483.2001. Mol Cell Biol. 2001. PMID: 11533236 Free PMC article.

3

The t(8;21) fusion protein, AML1 ETO, specifically represses the transcription of the p14(ARF) tumor suppressor in acute myeloid leukemia.

Linggi B, Müller-Tidow C, van de Locht L, Hu M, Nip J, Serve H, Berdel WE, van der Reijden B, Quelle DE, Rowley JD, Cleveland J, Jansen JH, Pandolfi PP, Hiebert SW. Linggi B, et al. Among authors: nip j. Nat Med. 2002 Jul;8(7):743-50. doi: 10.1038/nm726. Epub 2002 Jun 24. Nat Med. 2002. PMID: 12091906

4

E2F-1 cooperates with topoisomerase II inhibition and DNA damage to selectively augment p53-independent apoptosis.

Nip J, Strom DK, Fee BE, Zambetti G, Cleveland JL, Hiebert SW. Nip J, et al. Mol Cell Biol. 1997 Mar;17(3):1049-56. doi: 10.1128/MCB.17.3.1049. Mol Cell Biol. 1997. PMID: 9032231 Free PMC article.

5

E2F-1 and E2F-3 are functionally distinct in their ability to promote myeloid cell cycle progression and block granulocyte differentiation.

Strom DK, Cleveland JL, Chellappan S, Nip J, Hiebert SW. Strom DK, et al. Among authors: nip j. Cell Growth Differ. 1998 Jan;9(1):59-69. Cell Growth Differ. 1998. PMID: 9438389

6

Expression of the AML-1 oncogene shortens the G(1) phase of the cell cycle.

Strom DK, Nip J, Westendorf JJ, Linggi B, Lutterbach B, Downing JR, Lenny N, Hiebert SW. Strom DK, et al. Among authors: nip j. J Biol Chem. 2000 Feb 4;275(5):3438-45. doi: 10.1074/jbc.275.5.3438. J Biol Chem. 2000. PMID: 10652337 Free article.

7

E2F-1 induces the stabilization of p53 but blocks p53-mediated transactivation.

Nip J, Strom DK, Eischen CM, Cleveland JL, Zambetti GP, Hiebert SW. Nip J, et al. Oncogene. 2001 Feb 22;20(8):910-20. doi: 10.1038/sj.onc.1204171. Oncogene. 2001. PMID: 11314026

8

Topoisomerase IIalpha mediates E2F-1-induced chemosensitivity and is a target for p53-mediated transcriptional repression.

Nip J, Hiebert SW. Nip J, et al. Cell Biochem Biophys. 2000;33(2):199-207. doi: 10.1385/CBB:33:2:199. Cell Biochem Biophys. 2000. PMID: 11325040

9

Bcl-2 is an apoptotic target suppressed by both c-Myc and E2F-1.

Eischen CM, Packham G, Nip J, Fee BE, Hiebert SW, Zambetti GP, Cleveland JL. Eischen CM, et al. Among authors: nip j. Oncogene. 2001 Oct 25;20(48):6983-93. doi: 10.1038/sj.onc.1204892. Oncogene. 2001. PMID: 11704823

10

Paracrine growth stimulation by hepatocyte-derived insulin-like growth factor-1: a regulatory mechanism for carcinoma cells metastatic to the liver.

Long L, Nip J, Brodt P. Long L, et al. Among authors: nip j. Cancer Res. 1994 Jul 15;54(14):3732-7. Cancer Res. 1994. PMID: 7518344

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