Ko KH - Search Results

1

Effect of repeated seizure experiences on tyrosine hydroxylase immunoreactivities in the brain of genetically epilepsy-prone rats.

Ryu JR, Shin CY, Park KH, Jeon GS, Kim H, Kim W, Dailey JW, Jobe PC, Cho SS, Ko KH. Ryu JR, et al. Among authors: ko kh. Brain Res Bull. 2000 Dec;53(6):777-82. doi: 10.1016/s0361-9230(00)00373-7. Brain Res Bull. 2000. PMID: 11179842

2

Noradrenergic abnormalities in the genetically epilepsy-prone rat.

Jobe PC, Mishra PK, Browning RA, Wang C, Adams-Curtis LE, Ko KH, Dailey JW. Jobe PC, et al. Among authors: ko kh. Brain Res Bull. 1994;35(5-6):493-504. doi: 10.1016/0361-9230(94)90163-5. Brain Res Bull. 1994. PMID: 7859107 Review.

3

Morphological deficits in noradrenergic neurons in GEPR-9s stem from abnormalities in both the locus coeruleus and its target tissues.

Ryu JR, Jobe PC, Milbrandt JC, Mishra PK, Clough RW, Browning RA, Dailey JW, Seo DO, Ko KH. Ryu JR, et al. Among authors: ko kh. Exp Neurol. 1999 Mar;156(1):84-91. doi: 10.1006/exnr.1998.7003. Exp Neurol. 1999. PMID: 10192779

4

Effect of norepinephrine release on adrenoceptors in severe seizure genetically epilepsy-prone rats.

Seo DO, Shin CY, Ryu JR, Cheong JH, Choi CR, Dailey JW, Reith ME, Jobe PC, Ko KH. Seo DO, et al. Among authors: ko kh. Eur J Pharmacol. 2000 May 19;396(2-3):53-8. doi: 10.1016/s0014-2999(00)00225-9. Eur J Pharmacol. 2000. PMID: 10822056

5

Immunostimulation of rat primary astrocytes decreases intracellular ATP level.

Shin CY, Choi JW, Ryu JR, Ryu JH, Kim W, Kim H, Ko KH. Shin CY, et al. Among authors: ko kh. Brain Res. 2001 Jun 1;902(2):198-204. doi: 10.1016/s0006-8993(01)02385-x. Brain Res. 2001. PMID: 11384613

6

Glucocorticoids exacerbate peroxynitrite mediated potentiation of glucose deprivation-induced death of rat primary astrocytes.

Shin CY, Choi JW, Jang ES, Ryu JH, Kim WK, Kim HC, Ko KH. Shin CY, et al. Among authors: ko kh. Brain Res. 2001 Dec 27;923(1-2):163-71. doi: 10.1016/s0006-8993(01)03212-7. Brain Res. 2001. PMID: 11743984

7

Abnormalities in norepinephrine turnover rate in the central nervous system of the genetically epilepsy-prone rat.

Jobe PC, Ko KH, Dailey JW. Jobe PC, et al. Among authors: ko kh. Brain Res. 1984 Jan 9;290(2):357-60. doi: 10.1016/0006-8993(84)90956-9. Brain Res. 1984. PMID: 6692149

8

Decrease in the kainate-induced wet dog shake behavior in genetically epilepsy-prone rats: possible involvement of an impaired synaptic transmission to the 5-HT(2A) receptor.

Shin EJ, Jeong JH, Chung YH, Kim TW, Shin CY, Kim WK, Ko KH, Kim HC. Shin EJ, et al. Among authors: ko kh. J Pharmacol Sci. 2009 Jul;110(3):401-4. doi: 10.1254/jphs.09015sc. J Pharmacol Sci. 2009. PMID: 19609070 Free article.

9

Oxidative damage causes formation of lipofuscin-like substances in the hippocampus of the senescence-accelerated mouse after kainate treatment.

Kim HC, Bing G, Jhoo WK, Kim WK, Shin EJ, Park ES, Choi YS, Lee DW, Shin CY, Ryu JR, Ko KH. Kim HC, et al. Among authors: ko kh. Behav Brain Res. 2002 Apr 1;131(1-2):211-20. doi: 10.1016/s0166-4328(01)00382-5. Behav Brain Res. 2002. PMID: 11844588

10

Evidence that protease-activated receptor-2 mediates trypsin-induced reversal of stellation in cultured rat astrocytes.

Park GH, Ryu JR, Shin CY, Choi MS, Han BH, Kim WK, Kim HC, Ko KH. Park GH, et al. Among authors: ko kh. Neurosci Res. 2006 Jan;54(1):15-23. doi: 10.1016/j.neures.2005.09.007. Epub 2005 Oct 26. Neurosci Res. 2006. PMID: 16256233

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