Palotás A - Search Results

1

Comparative studies on [Ca2+]i-level of fibroblasts from Alzheimer patients and control individuals.

Palotás A, Kálmán J, Laskay G, Juhász A, Janka Z, Penke B. Palotás A, et al. Neurochem Res. 2001 Jul;26(7):817-20. doi: 10.1023/a:1011664102849. Neurochem Res. 2001. PMID: 11565613

2

[Change of fibroblast calcium levels caused by beta-amyloid peptide in Alzheimer disease].

Palotás A, Kálmán J, Laskay G, Juhász A, Janka Z, Penke B. Palotás A, et al. Ideggyogy Sz. 2002 May 20;55(5-6):164-7. Ideggyogy Sz. 2002. PMID: 12122875 Hungarian.

3

Beta-amyloid-induced increase in the resting intracellular calcium concentration gives support to tell Alzheimer lymphocytes from control ones.

Palotás A, Kálmán J, Palotás M, Juhász A, Janka Z, Penke B. Palotás A, et al. Among authors: palotas m. Brain Res Bull. 2002 Jun;58(2):203-5. doi: 10.1016/s0361-9230(02)00773-6. Brain Res Bull. 2002. PMID: 12127018

4

Fibroblasts and lymphocytes from Alzheimer patients are resistant to beta-amyloid-induced increase in the intracellular calcium concentration.

Palotás A, Kálmán J, Palotás M, Juhász A, Janka Z, Penke B. Palotás A, et al. Among authors: palotas m. Prog Neuropsychopharmacol Biol Psychiatry. 2002 Jun;26(5):971-4. doi: 10.1016/s0278-5846(02)00214-2. Prog Neuropsychopharmacol Biol Psychiatry. 2002. PMID: 12369273

5

Whole blood samples from Alzheimer patients and control donors demonstrate fluorimetric differences.

Palotás A, Kálmán J, Palotás M, Matin K, Szentpáli K, Paszt A, Janka Z, Penke B. Palotás A, et al. Among authors: palotas m. Neurochem Res. 2002 Dec;27(12):1589-91. doi: 10.1023/a:1021618523577. Neurochem Res. 2002. PMID: 12515309

6

Long-term exposition of cells to beta-amyloid results in decreased intracellular calcium concentration.

Palotás A, Kálmán J, Palotás M, Kemény L, Janka Z, Penke B. Palotás A, et al. Among authors: palotas m. Neurochem Int. 2003 Jun;42(7):543-7. doi: 10.1016/s0197-0186(02)00188-2. Neurochem Int. 2003. PMID: 12590936

7

The effect of citalopram on gene expression profile of Alzheimer lymphocytes.

Palotás A, Puskás LG, Kitajka K, Palotás M, Molnár J, Pákáski M, Janka Z, Penke B, Kálmán J. Palotás A, et al. Among authors: palotas m. Neurochem Res. 2004 Aug;29(8):1563-70. doi: 10.1023/b:nere.0000029570.57903.74. Neurochem Res. 2004. PMID: 15260135

8

ApoE -491A/T promoter polymorphism is not an independent risk factor, but associated with the epsilon4 allele in Hungarian Alzheimer's dementia population.

Juhász A, Palotás A, Janka Z, Rimanóczy A, Palotás M, Bódi N, Boda K, Zana M, Vincze G, Kálmán J. Juhász A, et al. Among authors: palotas m, palotas a. Neurochem Res. 2005 May;30(5):591-6. doi: 10.1007/s11064-005-2745-6. Neurochem Res. 2005. PMID: 16176061

9

CYP46 T/C polymorphism is not associated with Alzheimer's dementia in a population from Hungary.

Juhász A, Rimanóczy A, Boda K, Vincze G, Szlávik G, Zana M, Bjelik A, Pákáski M, Bódi N, Palotás A, Janka Z, Kálmán J. Juhász A, et al. Among authors: palotas a. Neurochem Res. 2005 Aug;30(8):943-8. doi: 10.1007/s11064-005-5979-4. Neurochem Res. 2005. PMID: 16258842

10

Haloperidol attenuates beta-amyloid-induced calcium imbalance in human fibroblasts.

Palotás A, Penke B, Palotás M, Kenderessy AS, Kemény L, Kis E, Vincze G, Janka Z, Kálmán J. Palotás A, et al. Among authors: palotas m. Skin Pharmacol Physiol. 2004 Jul-Aug;17(4):195-9. doi: 10.1159/000078823. Skin Pharmacol Physiol. 2004. PMID: 15258451

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