Hagglund R - Search Results

1

Herpes simplex virus 1 mutant in which the ICP0 HUL-1 E3 ubiquitin ligase site is disrupted stabilizes cdc34 but degrades D-type cyclins and exhibits diminished neurotoxicity.

Hagglund R, Roizman B. Hagglund R, et al. J Virol. 2003 Dec;77(24):13194-202. doi: 10.1128/jvi.77.24.13194-13202.2003. J Virol. 2003. PMID: 14645576 Free PMC article.

2

U(S)3 protein kinase of herpes simplex virus 1 blocks caspase 3 activation induced by the products of U(S)1.5 and U(L)13 genes and modulates expression of transduced U(S)1.5 open reading frame in a cell type-specific manner.

Hagglund R, Munger J, Poon AP, Roizman B. Hagglund R, et al. J Virol. 2002 Jan;76(2):743-54. doi: 10.1128/jvi.76.2.743-754.2002. J Virol. 2002. PMID: 11752164 Free PMC article.

3

Herpes simplex virus 1-infected cell protein 0 contains two E3 ubiquitin ligase sites specific for different E2 ubiquitin-conjugating enzymes.

Hagglund R, Van Sant C, Lopez P, Roizman B. Hagglund R, et al. Proc Natl Acad Sci U S A. 2002 Jan 22;99(2):631-6. doi: 10.1073/pnas.022531599. Proc Natl Acad Sci U S A. 2002. PMID: 11805320 Free PMC article.

4

Characterization of the novel E3 ubiquitin ligase encoded in exon 3 of herpes simplex virus-1-infected cell protein 0.

Hagglund R, Roizman B. Hagglund R, et al. Proc Natl Acad Sci U S A. 2002 Jun 11;99(12):7889-94. doi: 10.1073/pnas.122246999. Proc Natl Acad Sci U S A. 2002. PMID: 12060736 Free PMC article.

5

Infected cell protein No. 22 is subject to proteolytic cleavage by caspases activated by a mutant that induces apoptosis.

Munger J, Hagglund R, Roizman B. Munger J, et al. Among authors: hagglund r. Virology. 2003 Jan 20;305(2):364-70. doi: 10.1006/viro.2002.1728. Virology. 2003. PMID: 12573581 Free article.

6

Role of ICP0 in the strategy of conquest of the host cell by herpes simplex virus 1.

Hagglund R, Roizman B. Hagglund R, et al. J Virol. 2004 Mar;78(5):2169-78. doi: 10.1128/jvi.78.5.2169-2178.2004. J Virol. 2004. PMID: 14963113 Free PMC article. Review. No abstract available.

7

The infected cell protein 0 of herpes simplex virus 1 dynamically interacts with proteasomes, binds and activates the cdc34 E2 ubiquitin-conjugating enzyme, and possesses in vitro E3 ubiquitin ligase activity.

Van Sant C, Hagglund R, Lopez P, Roizman B. Van Sant C, et al. Among authors: hagglund r. Proc Natl Acad Sci U S A. 2001 Jul 17;98(15):8815-20. doi: 10.1073/pnas.161283098. Epub 2001 Jul 10. Proc Natl Acad Sci U S A. 2001. PMID: 11447293 Free PMC article.

8

Posttranslational processing of infected cell proteins 0 and 4 of herpes simplex virus 1 is sequential and reflects the subcellular compartment in which the proteins localize.

Advani SJ, Hagglund R, Weichselbaum RR, Roizman B. Advani SJ, et al. Among authors: hagglund r. J Virol. 2001 Sep;75(17):7904-12. doi: 10.1128/jvi.75.17.7904-7912.2001. J Virol. 2001. PMID: 11483735 Free PMC article.

9

Paramyxovirus fusion (F) protein: a conformational change on cleavage activation.

Dutch RE, Hagglund RN, Nagel MA, Paterson RG, Lamb RA. Dutch RE, et al. Among authors: hagglund rn. Virology. 2001 Mar 1;281(1):138-50. doi: 10.1006/viro.2000.0817. Virology. 2001. PMID: 11222104 Free article.

10

The social implications of medical technology.

Hagglund RA. Hagglund RA. Med Instrum. 1979 Jan-Feb;13(1):21-3. Med Instrum. 1979. PMID: 106228 No abstract available.

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